Interactions between genetic polymorphisms in the apoptotic pathway and environmental factors on esophageal adenocarcinoma risk

Carcinogenesis

Volumn 32, Issue 4, 2011, Pages 502-506

  Wu, I Chen ^a,b   Zhao, Yang ^a   Zhai, Rihong ^a   Liu, Chen Yu ^a   Chen, Feng ^a   Ter Minassian, Monica ^a   Asomaning, Kofi ^a   Su, Li ^a   Heist, Rebecca S ^a,c,d   Kulke, Matthew H ^e   Liu, Geoffrey ^a,f   Christiani, David C ^a,c,d  

^a HARVARD SCHOOL OF PUBLIC HEALTH   (United States)

^b KAOHSIUNG MEDICAL UNIVERSITY HOSPITAL   (Taiwan)

^c MASSACHUSETTS GENERAL HOSPITAL   (United States)

^d HARVARD MEDICAL SCHOOL   (United States)

^e DANA FARBER CANCER INSTITUTE   (United States)

^f PRINCESS MARGARET HOSPITAL   (Canada)

Author keywords

[No Author keywords available]

Indexed keywords

ADULT; ALGORITHM; ALLELE; APOPTOSIS; ARTICLE; BODY MASS; CANCER PATIENT; CANCER RISK; CARCINOGENESIS; CAUCASIAN; ENVIRONMENTAL FACTOR; ESOPHAGEAL ADENOCARCINOMA; FEMALE; GASTROESOPHAGEAL REFLUX; GENE INTERACTION; GENETIC POLYMORPHISM; GENETIC VARIABILITY; GENOTYPE; HUMAN; MAJOR CLINICAL STUDY; MALE; PRIORITY JOURNAL; RISK ASSESSMENT; SINGLE NUCLEOTIDE POLYMORPHISM; SMOKING;

ADENOCARCINOMA; ADULT; AGED; APOPTOSIS; CASPASE 7; ESOPHAGEAL NEOPLASMS; FEMALE; GASTROESOPHAGEAL REFLUX; HUMANS; LOGISTIC MODELS; MALE; MIDDLE AGED; PHOSPHATIDYLINOSITOL 3-KINASES; POLYMORPHISM, SINGLE NUCLEOTIDE; PROTO-ONCOGENE PROTEINS C-AKT; RECEPTORS, TUMOR NECROSIS FACTOR, TYPE I; RISK; SMOKING;

EID: 79953676585     PISSN: 01433334     EISSN: 14602180     Source Type: Journal    
DOI: 10.1093/carcin/bgq287     Document Type: Article

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