FLIP regulation of HO-1 and TNF signalling in human acute myeloid leukemia provides a unique secondary anti-apoptotic mechanism.

Oncotarget

Volumn 1, Issue 5, 2010, Pages 359-366

  Rushworth, Stuart A ^a   Zaitseva, Lyubov ^b   Langa, Susana ^b   Bowles, Kristian M ^b   MacEwan, David J ^b  

^a UNIVERSITY OF EAST ANGLIA   (United Kingdom)

^b NONE

Author keywords

[No Author keywords available]

Indexed keywords

ANTINEOPLASTIC AGENT; FLICE INHIBITORY PROTEIN; HEME OXYGENASE 1; HMOX1 PROTEIN, HUMAN; IMMUNOGLOBULIN ENHANCER BINDING PROTEIN; MESSENGER RNA; TUMOR NECROSIS FACTOR ALPHA;

ACUTE GRANULOCYTIC LEUKEMIA; APOPTOSIS; ARTICLE; CELL STRAIN HL 60; DRUG EFFECT; DRUG RESISTANCE; ENZYMOLOGY; GENETICS; HUMAN; METABOLISM; PATHOLOGY; RNA INTERFERENCE; SIGNAL TRANSDUCTION; TIME; TUMOR CELL CULTURE;

ANTINEOPLASTIC AGENTS; APOPTOSIS; CASP8 AND FADD-LIKE APOPTOSIS REGULATING PROTEIN; DRUG RESISTANCE, NEOPLASM; HEME OXYGENASE-1; HL-60 CELLS; HUMANS; LEUKEMIA, MYELOID, ACUTE; NF-KAPPA B; RNA INTERFERENCE; RNA, MESSENGER; SIGNAL TRANSDUCTION; TIME FACTORS; TUMOR CELLS, CULTURED; TUMOR NECROSIS FACTOR-ALPHA;

EID: 79953665817     PISSN: None     EISSN: 19492553     Source Type: Journal    
DOI: 10.18632/oncotarget.168     Document Type: Article

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