Pathophysiologic bases of medical therapies for endometriosis

Sexuality, Reproduction and Menopause

Volumn 9, Issue 1, 2011, Pages 31-33

  Giudice, Linda C ^a  

^a UNIVERSITY OF CALIFORNIA   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ANDROGEN; ANGIOGENESIS INHIBITOR; ANTIESTROGEN; ANTIGESTAGEN; AROMATASE INHIBITOR; DANAZOL; GESTAGEN; GONADORELIN AGONIST; GONADORELIN ANTAGONIST; INTERLEUKIN 1; INTERLEUKIN 8; LEVONORGESTREL; MATRIX METALLOPROTEINASE INHIBITOR; NERVE GROWTH FACTOR; NONSTEROID ANTIINFLAMMATORY AGENT; ORAL CONTRACEPTIVE AGENT; PEROXISOME PROLIFERATOR ACTIVATED RECEPTOR GAMMA AGONIST; STEROID; TUMOR NECROSIS FACTOR ALPHA; TUMOR NECROSIS FACTOR ALPHA INHIBITOR; VASCULOTROPIN;

ANTIANGIOGENIC ACTIVITY; ANTIINFLAMMATORY ACTIVITY; ANTIOXIDANT ACTIVITY; ARTICLE; DECIDUALIZATION; DRUG EFFICACY; ENDOMETRIOSIS; HERBAL MEDICINE; HUMAN; IMMUNOMODULATION; INFLAMMATION; LONG TERM CARE; NONHUMAN; PAIN; PATHOPHYSIOLOGY; PRIORITY JOURNAL; THERAPY EFFECT; UNSPECIFIED SIDE EFFECT; UTERUS SURGERY;

EID: 79953281166     PISSN: 15462501     EISSN: 18787576     Source Type: Journal    
DOI: None     Document Type: Article

References (7)

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2. Nayyar T, Bruner-Tran KL, Piestrzeniewicz-Ulanska D, et al. Developmental exposure of mice to TCDD elicits a similar uterine phenotype in adult animals as observed in women with endometriosis. Reprod Toxicol. 2007;23:326-336.
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7. Berkley KJ, Rapkin AJ, Papka RE. The pains of endometriosis. Science. 2005;308:1587-1589.