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Volumn 66, Issue 4, 2011, Pages 941-942
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Mutation Vif-22H, which allows HIV-1 to use the APOBEC3G hypermutation to develop resistance, could appear more quickly in certain non-B variants
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Author keywords
HIV drug resistance mutations; HIV 1 subtypes and recombinants; Mechanisms of resistance
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Indexed keywords
APOLIPOPROTEIN B MESSENGER RNA EDITING ENZYME CATALYTIC POLYPEPTIDE 3G;
ASPARAGINE;
HISTIDINE;
HUMAN IMMUNODEFICIENCY VIRUS PROTEINASE;
LYSINE;
RNA DIRECTED DNA POLYMERASE;
VIF PROTEIN;
ANTIVIRAL RESISTANCE;
CONTROLLED STUDY;
DNA POLYMORPHISM;
GENE SEQUENCE;
GENETIC VARIABILITY;
HUMAN IMMUNODEFICIENCY VIRUS 1;
LETTER;
NONHUMAN;
VIRUS GENE;
VIRUS MUTATION;
ADAPTATION, BIOLOGICAL;
CYTIDINE DEAMINASE;
DRUG RESISTANCE, VIRAL;
GENOTYPE;
HIV-1;
HUMANS;
MUTATION, MISSENSE;
RECOMBINATION, GENETIC;
VIF GENE PRODUCTS, HUMAN IMMUNODEFICIENCY VIRUS;
VIRULENCE FACTORS;
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EID: 79952802313
PISSN: 03057453
EISSN: 14602091
Source Type: Journal
DOI: 10.1093/jac/dkr012 Document Type: Letter |
Times cited : (7)
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References (6)
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