Dietary zinc supplementation of 3xTg-AD mice increases BDNF levels and prevents cognitive deficits as well as mitochondrial dysfunction.

Cell death & disease

Volumn 1, Issue , 2010, Pages

  Corona, C ^a   Masciopinto, F ^b   Silvestri, E ^b   Viscovo, A Del ^b   Lattanzio, R ^b   Sorda, R L ^b   Ciavardelli, D ^b   Goglia, F ^b   Piantelli, M ^b   Canzoniero, L M ^b   Sensi, S L ^b  

^a UNIVERSITY OF CHIETI   (Italy)

^b NONE

Author keywords

[No Author keywords available]

Indexed keywords

AMYLOID BETA PROTEIN; BRAIN DERIVED NEUROTROPHIC FACTOR; TAU PROTEIN; ZINC;

ALZHEIMER DISEASE; ANIMAL; ARTICLE; COGNITIVE DEFECT; DIET SUPPLEMENTATION; HIPPOCAMPUS; METABOLISM; MITOCHONDRION; MOUSE; PATHOLOGY; PHYSIOLOGY; TRANSGENIC MOUSE;

ALZHEIMER DISEASE; AMYLOID BETA-PEPTIDES; ANIMALS; BRAIN-DERIVED NEUROTROPHIC FACTOR; COGNITION DISORDERS; DIETARY SUPPLEMENTS; HIPPOCAMPUS; MICE; MICE, TRANSGENIC; MITOCHONDRIA; TAU PROTEINS; ZINC;

EID: 79952638895     PISSN: None     EISSN: 20414889     Source Type: Journal    
DOI: 10.1038/cddis.2010.73     Document Type: Article

References (0)