Pro-inflammatory angiogenesis is mediated by p38 MAP kinase.

Journal of cellular physiology

Volumn 226, Issue 3, 2011, Pages 800-808

  Rajashekhar, Gangaraju ^a   Kamocka, Malgorzata ^b   Marin, Abby ^b   Suckow, Mark A ^b   Wolter, William R ^b   Badve, Sunil ^b   Sanjeevaiah, Aravind Raj ^b   Pumiglia, Kevin ^b   Rosen, Elliot ^b   Clauss, Matthias ^b  

^a INDIANA UNIVERSITY SCHOOL OF MEDICINE   (United States)

^b NONE

Author keywords

[No Author keywords available]

Indexed keywords

GELATINASE B; HYDROGEN PEROXIDE; MITOGEN ACTIVATED PROTEIN KINASE P38; TUMOR NECROSIS FACTOR ALPHA;

ANIMAL; ARTICLE; BIOLOGICAL MODEL; CELL PROLIFERATION; DRUG ANTAGONISM; DRUG EFFECT; ENDOTHELIUM CELL; ENZYME ACTIVATION; ENZYMOLOGY; HUMAN; INFLAMMATION; METABOLISM; MOUSE; NEOPLASM; NEOVASCULARIZATION (PATHOLOGY); PATHOLOGY; RAT; SIGNAL TRANSDUCTION; SOLUBILITY; VASCULARIZATION;

ANIMALS; CELL PROLIFERATION; ENDOTHELIAL CELLS; ENZYME ACTIVATION; HUMANS; HYDROGEN PEROXIDE; INFLAMMATION; MAP KINASE SIGNALING SYSTEM; MATRIX METALLOPROTEINASE 9; MICE; MODELS, BIOLOGICAL; NEOPLASMS; NEOVASCULARIZATION, PATHOLOGIC; P38 MITOGEN-ACTIVATED PROTEIN KINASES; RATS; SOLUBILITY; TUMOR NECROSIS FACTOR-ALPHA;

EID: 79952116012     PISSN: None     EISSN: 10974652     Source Type: Journal    
DOI: 10.1002/jcp.22404     Document Type: Article

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