Isoniazid and rifampicin resistance-associated mutations in Mycobacterium tuberculosis isolates from Yangon, Myanmar: Implications for rapid molecular testing-authors' response

Journal of Antimicrobial Chemotherapy

Volumn 66, Issue 3, 2011, Pages 687-

  Syre, Heidi ^a,b   Grewal, Harleen ^a,b  

^a UNIVERSITY OF BERGEN   (Norway)

^b HAUKELAND UNIVERSITY HOSPITAL   (Norway)

Author keywords

Drug susceptibility; Molecular characterization; TB

Indexed keywords

ISONIAZID; RIFAMPICIN;

ANTIBIOTIC RESISTANCE; BACTERIAL MUTATION; BACTERIAL STRAIN; BACTERIUM ISOLATE; DNA DETERMINATION; LETTER; MYANMAR; MYCOBACTERIUM TUBERCULOSIS; NONHUMAN; PHYLOGENY; PSEUDOGENE; SINGLE NUCLEOTIDE POLYMORPHISM;

EID: 79951527314     PISSN: 03057453     EISSN: 14602091     Source Type: Journal    
DOI: 10.1093/jac/dkq514     Document Type: Letter

References (4)

1. Köser CU, Summers DK, Archer JAC. Comment on: Isoniazid and rifampicin resistance-associated mutations in Mycobacterium tuberculosis isolates from Yangon, Myanmar: implications for rapid molecular testing. J Antimicrob Chemother 2011; 66: 686-7.
2. Valvatne H, Syre H, Kross M et al. Isoniazid and rifampicin resistance-associated mutations in Mycobacterium tuberculosis isolates from Yangon, Myanmar: implications for rapid molecular testing. J Antimicrob Chemother 2009; 64: 694-701.
3. -) type. PLoS ONE 2009; 4: e4540.
4. Baker VL, Brown TJ, Maxwell O et al. Molecular analysis of isoniazid-resistant Mycobacterium tuberculosis isolates from England and Wales reveals the phylogenetic significance of the ahpC -46A polymorphism. Antimicrob Agents Chemother 2005; 49: 1455-64.