Functional effects of a tropomyosin mutation linked to FHC contribute to maladaptation during acidosis

Journal of Molecular and Cellular Cardiology

Volumn 50, Issue 3, 2011, Pages 442-450

  Sheehan, Katherine A ^a   Arteaga, Grace M ^c   Hinken, Aaron C ^a   Dias, Fernando A ^a   Ribeiro, Cibele ^a   Wieczorek, David F ^b   Solaro, R John ^a   Wolska, Beata M ^a  

^a UNIVERSITY OF ILLINOIS AT CHICAGO   (United States)

^b UNIVERSITY OF CINCINNATI COLLEGE OF MEDICINE   (United States)

^c MAYO CLINIC   (United States)

Author keywords

Acidosis; Calcium; Contractility; FHC; Myofilament calcium sensitivity; Tropomyosin

Indexed keywords

CALCIUM ION; TROPOMYOSIN;

ACIDITY; ACIDOSIS; ANIMAL CELL; ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; FAMILIAL HYPERTROPHIC CARDIOMYOPATHY; GENE MUTATION; HEART MUSCLE CELL; HEART MUSCLE CONTRACTILE FORCE; HEART MUSCLE RELAXATION; HEART MUSCLE TENSION; HEART PAPILLARY MUSCLE; MOUSE; MUSCLE ISOMETRIC CONTRACTION; MYOFILAMENT; NONHUMAN; PRIORITY JOURNAL; PROTEIN PHOSPHORYLATION; TRANSGENIC MOUSE;

ACIDOSIS; ANIMALS; CALCIUM; CARDIOMYOPATHY, HYPERTROPHIC, FAMILIAL; CELLS, CULTURED; DEATH, SUDDEN, CARDIAC; ISOMETRIC CONTRACTION; MICE; MICE, TRANSGENIC; MICROFILAMENTS; MUTATION; MYOCARDIAL CONTRACTION; MYOCYTES, CARDIAC; PAPILLARY MUSCLES; PHOSPHORYLATION; TROPOMYOSIN;

MUS; MUS MUSCULUS;

EID: 79551611313     PISSN: 00222828     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.yjmcc.2010.10.032     Document Type: Article

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