The Kidney is not always the target in neurogenic hypertension

Journal of Applied Physiology

Volumn 109, Issue 6, 2010, Pages 2008-

  Osborn, John W ^a   Fink, Gregory D ^a  

^a UNIVERSITY OF MINNESOTA   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ADRENERGIC SYSTEM; ANIMAL; BLOOD PRESSURE; HUMAN; HYPERTENSION; INNERVATION; KIDNEY; METABOLISM; NOTE; PATHOPHYSIOLOGY; RENIN ANGIOTENSIN ALDOSTERONE SYSTEM; SYMPATHECTOMY;

ANIMALS; BLOOD PRESSURE; HUMANS; HYPERTENSION; KIDNEY; RENIN-ANGIOTENSIN SYSTEM; SYMPATHECTOMY; SYMPATHETIC NERVOUS SYSTEM;

EID: 78651274858     PISSN: 87507587     EISSN: 15221601     Source Type: Journal    
DOI: 10.1152/japplphysiol.01160.2010     Document Type: Note

References (6)

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2. King AJ, Osborn JW, Fink GD. Splanchnic circulation is a critical neural target in angiotensin II salt hypertension in rats. Hypertension 50:547-556, 2007.
3. Luft FC, Wilcox CS, Unger T, Kuhn R, Demmert G, Rohmeiss P, Ganten D, Sterzel RB. Angiotensin-induced hypertension in the rat: sympathetic nerve activity and prostaglandins. Hypertension 14:396-403, 1989.
4. Moretti JL, Burke SL, Evans RG, Lambert GW, Head GA. Enhanced responses to ganglion blockade do not reflect sympathetic nervous system contribution to Ang II-induced hypertension. J Hypertens 27:1838-1848, 2009.
5. Navar LG. Counterpoint: Activation of the intrarenal renin-angiotensin system is the dominant contributor to systemic hypertension. J Appl Physiol; doi: 10.1152/japplphysiol.00182.2010a.
6. Yoshimoto M, Miki K, Fink GD, King A, Osborn JW. Chronic angiotensin II infusion causes differential responses in regional sympathetic nerve activity in rats. Hypertension 55:644-651, 2010.