Complement receptor 3 (CD11b/CD18) is implicated in the elimination of β-amyloid peptides

Fundamental and Clinical Pharmacology

Volumn 25, Issue 1, 2011, Pages 115-122

  Choucair Jaafar, Nada ^a   Laporte, Vincent ^b   Levy, Rachel ^a   Poindron, Philippe ^a   Lombard, Yves ^a   Gies, Jean Pierre ^a  

^a UNIVERSITÉ DE STRASBOURG   (France)

^b UNIVERSITÉ CATHOLIQUE DE LOUVAIN   (Belgium)

Author keywords

amyloid peptide; Alzheimer's disease; Antibodies; Complement receptor; Low density lipoprotein receptor related protein; Microglia; Phagocytosis

Indexed keywords

AMYLOID BETA PROTEIN; CD11B ANTIGEN; CD18 ANTIGEN; COMPLEMENT RECEPTOR; LOW DENSITY LIPOPROTEIN RECEPTOR RELATED PROTEIN;

ARTICLE; CELL INTERACTION; COMPLEMENT ACTIVATION; INTERNALIZATION; MICROGLIA; NONHUMAN; PHAGOCYTOSIS; PRIORITY JOURNAL;

AMYLOID BETA-PEPTIDES; ANIMALS; ANTIGENS, CD11B; ANTIGENS, CD18; CELL LINE; LAMINARIA; LDL-RECEPTOR RELATED PROTEIN 1; MACROPHAGE-1 ANTIGEN; MICE; MICE, INBRED BALB C; MICROGLIA; PEPTIDE FRAGMENTS; SACCHAROMYCES CEREVISIAE;

EID: 78650940497     PISSN: 07673981     EISSN: 14728206     Source Type: Journal    
DOI: 10.1111/j.1472-8206.2010.00811.x     Document Type: Article

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