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Volumn 95, Issue 12, 2010, Pages

The pharmacological characteristics of molecular-based inherited salt-losing tubulopathies

(19)  Nozu, Kandai a   Iijima, Kazumoto a   Kanda, Kyoko a   Nakanishi, Koichi b   Yoshikawa, Norishige b   Satomura, Kenichi c   Kaito, Hiroshi a   Hashimura, Yuya a   Ninchoji, Takeshi a   Komatsu, Hiroshi d   Kamei, Koichi e   Miyashita, Ritsuko f   Kugo, Masaaki g   Ohashi, Hiroshi h   Yamazaki, Hajime i   Mabe, Hiroyo j   Otsubo, Asa k   Igarashi, Takashi l   Matsuo, Masafumi a  

d Japan   (Japan)

Author keywords

[No Author keywords available]

Indexed keywords

ATP SENSITIVE INWARD RECTIFIER POTASSIUM CHANNEL 1; CHLORIDE CHANNEL KB PROTEIN; FUROSEMIDE; MEMBRANE PROTEIN; SODIUM CHLORIDE COTRANSPORTER; SOLUTE CARRIER FAMILY 12 MEMBER 1; THIAZIDE DIURETIC AGENT; UNCLASSIFIED DRUG;

EID: 78650060800     PISSN: 0021972X     EISSN: 0021972X     Source Type: Journal    
DOI: 10.1210/jc.2010-0392     Document Type: Article
Times cited : (60)

References (21)
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    • Possible discrimination of Gitelman's syndrome from Bartter's syndrome by renal clearance study: Report of two cases
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    • Köckerling A, Reinalter SC, Seyberth HW 1996 Impaired response to furosemide in hyperprostaglandin E syndrome: evidence for a tubular defect in the loop of Henle. J Pediatr 129:519-528
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    • Novel mutations of the chloride channel Kb gene in two Japanese patients clinically diagnosed as bartter syndrome with hypocalciuria
    • DOI 10.1210/jc.2004-0775
    • Fukuyama S, Hiramatsu M, Akagi M, Higa M, Ohta T 2004 Novel mutations of the chloride channel Kb gene in two Japanese patients clinically diagnosed as Bartter syndrome with hypocalciuria. J Clin Endocrinol Metab 89:5847-5850 (Pubitemid 39518480)
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* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.