Staphylococcal-mediated worsening of atopic dermatitis: Many players involved

British Journal of Dermatology

Volumn 163, Issue 6, 2010, Pages 1147-

  Agner, T ^a  

^a BISPEBJERG HOSPITAL   (Denmark)

Author keywords

[No Author keywords available]

Indexed keywords

FILAGGRIN; SUPERANTIGEN;

ANTIGEN EXPRESSION; ATOPIC DERMATITIS; DISEASE SEVERITY; GENETIC ASSOCIATION; HUMAN; INFLAMMATION; LOSS OF FUNCTION MUTATION; NOTE; PRIORITY JOURNAL; SKIN WATER LOSS; STAPHYLOCOCCUS AUREUS;

CHILD; DERMATITIS, ATOPIC; HUMANS; INTERMEDIATE FILAMENT PROTEINS; STAPHYLOCOCCAL INFECTIONS; STAPHYLOCOCCUS AUREUS; SUPERANTIGENS;

EID: 78649669072     PISSN: 00070963     EISSN: 13652133     Source Type: Journal    
DOI: 10.1111/j.1365-2133.2010.10114.x     Document Type: Note

References (4)

1. Kozman A, Yao Y, Bina P, et al. Encoding a superantigen by Staphylococcus aureus does not affect clinical characteristics of infected atopic dermatitis lesions. Br J Dermatol 2010; 163: 1308-11.
2. Rodriguez E, Baurecht H, Herberich E, et al. Meta-analysis of filaggrin polymorphisms in eczema and asthma: robust risk factors in atopic disease. J Allergy Clin Immunol 2009; 123: 1361-70.
3. Ong PY, Ohtake T, Brandt C, et al. Endogenous antimicrobial peptides and skin infections in atopic dermatitis. N Engl J Med 2002; 347: 1151-60.
4. Howell MD, Fairchild HR, Kim BE, et al. Th2 cytokines act on S100/A11 to downregulate keratinocyte differentiation. J Invest Dermatol 2008; 128: 2248-58.