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AML1, the target of multiple chromosomal translocations in human leukemia, is essential for normal fetal liver hematopoiesis
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Mutations of AML1 are common in therapy-related myelodysplasia following therapy with alkylating agents and are significantly associated with deletion or loss of chromosome arm 7q and with subsequent leukemic transformation
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Point mutations in the RUNX1/AML1 gene: Another actor in RUNX leukemia
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Involvement of the AML1 gene in the t(3;21) in therapy-related leukemia and in chronic myeloid leukemia in blast crisis
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Consistent intergenic splicing and production of multiple transcripts between AML1 at 21q22 and unrelated genes at 3q26 in (3;21)(q26;q22) translocations
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Translocation 3;21, trisomy 8, and duplication of the Philadelphia chromosome: A rare but recurrent cytogenetic pathway in the blastic phase of chronic myeloid leukemia
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