Excessive apoptosis as a downstream molecular event during the development of congenital heart diseases

Circulation Journal

Volumn 74, Issue 11, 2010, Pages 2297-2298

  Kaichi, Shinji ^a,b   Hasegawa, Koji ^b   Morimoto, Tatsuya ^c  

^a KYOTO UNIVERSITY   (Japan)

^b NATIONAL HOSPITAL ORGANIZATION KYOTO MEDICAL CENTER   (Japan)

^c UNIVERSITY OF SHIZUOKA   (Japan)

Author keywords

[No Author keywords available]

Indexed keywords

ALPHA ACTIN; CASPASE 3; MYOSIN HEAVY CHAIN; PROTEIN BCL 2; T BOX TRANSCRIPTION FACTOR; TRANSCRIPTION FACTOR GATA 4; TRANSCRIPTION FACTOR NKX2.5; TRANSCRIPTION FACTOR TBX5;

APOPTOSIS; CONGENITAL HEART DISEASE; EDITORIAL; GENE MUTATION; HEART ATRIUM SEPTUM DEFECT; HEART MUSCLE CELL; HEART VENTRICLE SEPTUM DEFECT; HUMAN; NONHUMAN; PATHOGENESIS; PROTEIN EXPRESSION; SIGNAL TRANSDUCTION;

ACTINS; AGE FACTORS; ANIMALS; APOPTOSIS; APOPTOSIS REGULATORY PROTEINS; DOWN-REGULATION; GENE EXPRESSION REGULATION, DEVELOPMENTAL; GESTATIONAL AGE; HEART DEFECTS, CONGENITAL; HUMANS; MYOCYTES, CARDIAC; RNA, MESSENGER; TRANSCRIPTION FACTORS;

EID: 78349235801     PISSN: 13469843     EISSN: 13474820     Source Type: Journal    
DOI: 10.1253/circj.CJ-10-0930     Document Type: Editorial

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