Targeted disruption of K(ATP) channels aggravates cardiac toxicity in cocaine abuse.

Clinical and translational science

Volumn 2, Issue 5, 2009, Pages 361-365

  Reyes, Santiago ^a   Kane, Garvan C ^b   Zingman, Leonid V ^b   Yamada, Satsuki ^b   Terzic, Andre ^b  

^a MAYO CLINIC   (United States)

^b NONE

Author keywords

[No Author keywords available]

Indexed keywords

INWARDLY RECTIFYING POTASSIUM CHANNEL; INWARDLY RECTIFYING POTASSIUM CHANNEL SUBUNIT KIR6.2; POTASSIUM;

ANIMAL; ANIMAL EXPERIMENT; ARTICLE; BIOLOGICAL MODEL; C57BL MOUSE; CHEMISTRY; COCAINE DEPENDENCE; DISEASE MODEL; DRUG EFFECT; HEART; HEART MUSCLE CELL; HUMAN; HYPERTROPHY; METABOLISM; MOUSE; MOUSE MUTANT;

ANIMALS; COCAINE-RELATED DISORDERS; DISEASE MODELS, ANIMAL; HEART; HUMANS; HYPERTROPHY; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; MODELS, BIOLOGICAL; MYOCYTES, CARDIAC; PHYSICAL CONDITIONING, ANIMAL; POTASSIUM; POTASSIUM CHANNELS, INWARDLY RECTIFYING;

EID: 77957741647     PISSN: None     EISSN: 17528062     Source Type: Journal    
DOI: 10.1111/j.1752-8062.2009.00145.x     Document Type: Article

References (0)