Editorial: P53 in malignant glioma: 20 years later and still much to learn

Neuro-Oncology

Volumn 12, Issue 5, 2010, Pages 421-

  James, C David ^a  

^a NONE

Author keywords

[No Author keywords available]

Indexed keywords

CASPASE; CHLOROQUINE; PROTEIN P53;

ANTINEOPLASTIC ACTIVITY; APOPTOSIS; AUTOPHAGY; EDITORIAL; GLIOBLASTOMA; PROTEIN DEPLETION; PROTEIN EXPRESSION; PROTEIN FUNCTION; TUMOR SUPPRESSOR GENE; CELL DEATH; CELL PROLIFERATION; CELLULAR DISTRIBUTION; DRUG EFFICACY; GENE EXPRESSION; GENE TARGETING; HISTOPATHOLOGY; HUMAN; MEDIATOR; TREATMENT RESPONSE; TUMOR CELL LINE; TUMOR GROWTH; WILD TYPE;

EID: 77957063251     PISSN: 15228517     EISSN: 15235866     Source Type: Journal    
DOI: 10.1093/neuonc/noq037     Document Type: Editorial

References (2)

1. Geng Y, Kohli L, Klocke BJ, Roth KA. Chloroquine-induced autophagic vacuole accumulation and cell death in glioma cells is p53 independent. Neuro-Oncology 2010;12(5):473-481
2. Kim EL,Wüstenberg R, Rübsam A, Schmitz-Salue C,Warnecke G, Bücker EM, Pettkis N, Speidel D, RohdeV, Schulz-SchaefferW, DeppertW, Giese A. Chloroquine activates the p53 pathway and induces apoptosis in human glioma cells. Neuro-Oncology 2010;12(4):389-400