PI3K/Akt signaling is involved in the disruption of gap junctional communication caused by v-Src and TNF-α

Biochemical and Biophysical Research Communications

Volumn 400, Issue 2, 2010, Pages 230-235

  Ito, Satoko ^a   Hyodo, Toshinori ^a   Hasegawa, Hitoki ^a   Yuan, Hong ^a   Hamaguchi, Michinari ^a   Senga, Takeshi ^a  

^a NAGOYA UNIVERSITY GRADUATE SCHOOL OF MEDICINE   (Japan)

Author keywords

Akt; Connexin43; Gap junction; TNF ; V Src

Indexed keywords

CONNEXIN 43; INTERLEUKIN 1BETA; PHOSPHATIDYLINOSITOL 3 KINASE; PROTEIN AKT3; PROTEIN KINASE B; PROTEIN KINASE B BETA; PROTEIN SERINE THREONINE KINASE; PROTEIN TYROSINE KINASE; PROTEIN V SRC; TUMOR NECROSIS FACTOR ALPHA; UNCLASSIFIED DRUG; PROTEIN KINASE P60;

ANIMAL CELL; ARTICLE; CELL COMMUNICATION; CELL TRANSFORMATION; COMPETITIVE INHIBITION; CONTROLLED STUDY; FIBROBLAST; GAP JUNCTION; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROTEIN ANALYSIS; PROTEIN EXPRESSION; RAT; REGULATORY MECHANISM; SIGNAL TRANSDUCTION; ANIMAL; ENZYMOLOGY; GENETICS; METABOLISM; PHYSIOLOGY; TRANSFORMED CELL LINE;

1-PHOSPHATIDYLINOSITOL 3-KINASE; ANIMALS; CELL COMMUNICATION; CELL LINE, TRANSFORMED; GAP JUNCTIONS; MICE; ONCOGENE PROTEIN PP60(V-SRC); PROTO-ONCOGENE PROTEINS C-AKT; RATS; SIGNAL TRANSDUCTION; TUMOR NECROSIS FACTOR-ALPHA; PHOSPHATIDYLINOSITOL 3-KINASES;

EID: 77956781405     PISSN: 0006291X     EISSN: 10902104     Source Type: Journal    
DOI: 10.1016/j.bbrc.2010.08.045     Document Type: Article

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