Reduction of ICAM-1 expression by carbon monoxide via soluble guanylate cyclase activation accounts for modulation of neutrophil migration.

Naunyn-Schmiedeberg's archives of pharmacology

Volumn 381, Issue 6, 2010, Pages 483-493

  Dal Secco, Daniela ^a   Freitas, Andressa ^b   Abreu, Monica A ^b   Garlet, Thiago P ^b   Rossi, Marcos A ^b   Ferreira, Sérgio H ^b   Silva, João S ^b   Alves Filho, José C ^b   Cunha, Fernando Q ^b  

^a UNIVERSITY OF SÃO PAULO   (Brazil)

^b NONE

Author keywords

[No Author keywords available]

Indexed keywords

CARBON MONOXIDE; CD18 ANTIGEN; CELL RECEPTOR; ENZYME INHIBITOR; GUANYLATE CYCLASE; HEME OXYGENASE; INTERCELLULAR ADHESION MOLECULE 1; NITRIC OXIDE; PROTOPORPHYRIN; PROTOPORPHYRIN ZINC; SOLUBLE GUANYLYL CYCLASE;

ANIMAL; ARTICLE; C57BL MOUSE; CELL MOTION; DRUG ANTAGONISM; DRUG EFFECT; GENE EXPRESSION REGULATION; GENETICS; MALE; METABOLISM; MOUSE; MOUSE MUTANT; NEUTROPHIL; VASCULAR ENDOTHELIUM;

ANIMALS; ANTIGENS, CD18; CARBON MONOXIDE; CELL MOVEMENT; ENDOTHELIUM, VASCULAR; ENZYME INHIBITORS; GENE EXPRESSION REGULATION; GUANYLATE CYCLASE; HEME OXYGENASE (DECYCLIZING); INTERCELLULAR ADHESION MOLECULE-1; MALE; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; NEUTROPHILS; NITRIC OXIDE; PROTOPORPHYRINS; RECEPTORS, CYTOPLASMIC AND NUCLEAR;

EID: 77955869754     PISSN: None     EISSN: 14321912     Source Type: Journal    
DOI: 10.1007/s00210-010-0500-2     Document Type: Article

References (0)