Study on fingerprints correlated with pharmacodynamic of constituents in Herba Erigerontis against neurotoxicity induced by beta-amyloid peptide

Zhongguo Zhongyao Zazhi

Volumn 35, Issue 8, 2010, Pages 1038-1041

  Huang, Yong ^a   Qi, Xiaolan ^b   Guan, Zhizhong ^b   Wang, Yonglin ^a   Wang, Airain ^a   Li, Cuibing ^a   Chi, Mingyan ^a  

^a GUIZHOU MEDICAL UNIVERSITY   (China)

^b Key Laboratory of Molecular Biology   (China)

Author keywords

amyloid peptide; Alzheimer's disease; Constituents; Fingerprints and pharmacology; Herba Erigerontis

Indexed keywords

AMYLOID BETA PROTEIN; CHINESE DRUG; HERBACEOUS AGENT;

ALZHEIMER DISEASE; ARTICLE; CANCER CELL CULTURE; HERBA ERIGERONTIS; NEUROPROTECTION; NEUROTOXICITY; ULTRA PERFORMANCE LIQUID CHROMATOGRAPHY;

ALZHEIMER DISEASE; AMYLOID BETA-PROTEIN; ANALYSIS OF VARIANCE; ANIMALS; CELL LINE, TUMOR; CHROMATOGRAPHY, HIGH PRESSURE LIQUID; DRUGS, CHINESE HERBAL; ERIGERON; HUMANS; NERVOUS SYSTEM; NEUROTOXINS;

EID: 77955061025     PISSN: 10015302     EISSN: None     Source Type: Journal    
DOI: 10.4268/cjcmm20100822     Document Type: Article

References (10)

1. GuanZ Z, Miao H, Tian J Y, et al. Suppressed expression of nicotinic acetylcholine receptors by nanomolar beta-amyloid peptides in PC12 cells [J]. J Neural Transm, 2001,108:1417.