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Volumn 34, Issue 8, 2010, Pages

Successful treatment of imatinib-resistant hypereosinophilic syndrome with nilotinib

Author keywords

[No Author keywords available]

Indexed keywords

IMATINIB; NILOTINIB; PLATELET DERIVED GROWTH FACTOR ALPHA RECEPTOR; PREDNISONE;

EID: 77953628107     PISSN: 01452126     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.leukres.2010.02.024     Document Type: Letter
Times cited : (12)

References (7)
  • 1
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    • A tyrosine kinase created by fusion of the PDGFRA and FIP1L1 genes as a therapeutic target of imatinib in idiopathic hypereosinophilic syndrome
    • Cools J., DeAngelo D.J., Gotlib J., Stover E.H., Legare R.D., Cortes J., et al. A tyrosine kinase created by fusion of the PDGFRA and FIP1L1 genes as a therapeutic target of imatinib in idiopathic hypereosinophilic syndrome. N Engl J Med 2003, 348:1201-1214.
    • (2003) N Engl J Med , vol.348 , pp. 1201-1214
    • Cools, J.1    DeAngelo, D.J.2    Gotlib, J.3    Stover, E.H.4    Legare, R.D.5    Cortes, J.6
  • 2
    • 34248569784 scopus 로고    scopus 로고
    • Molecular mechanisms underlying FIP1L1-PDGFRA-mediated myeloproliferation
    • Buitenhuis M., Verhagen L.P., Cools J., Coffer P.J. Molecular mechanisms underlying FIP1L1-PDGFRA-mediated myeloproliferation. Cancer Res 2007, 67:3759-3766.
    • (2007) Cancer Res , vol.67 , pp. 3759-3766
    • Buitenhuis, M.1    Verhagen, L.P.2    Cools, J.3    Coffer, P.J.4
  • 3
    • 77749288978 scopus 로고    scopus 로고
    • Mechanisms of resistance to tyrosine kinase inhibitors in chronic myeloid leukemia and recent therapeutic strategies to overcome resistance
    • Bixby D., Talpaz M. Mechanisms of resistance to tyrosine kinase inhibitors in chronic myeloid leukemia and recent therapeutic strategies to overcome resistance. Hematol Am Soc Hematol Educ Prog 2009, 461-476.
    • (2009) Hematol Am Soc Hematol Educ Prog , pp. 461-476
    • Bixby, D.1    Talpaz, M.2
  • 4
    • 66849087150 scopus 로고    scopus 로고
    • FIP1L1-PDGFRalpha D842V, a novel pan resistant mutant, emerging after treatment of FIP1L1-PDGFRalpha T674I eosinophilic leukemia with single agent sorafenib
    • Lierman E., Michaux L., Beullens E., Pierre P., Marynen P., Cools J., et al. FIP1L1-PDGFRalpha D842V, a novel pan resistant mutant, emerging after treatment of FIP1L1-PDGFRalpha T674I eosinophilic leukemia with single agent sorafenib. Leukemia 2009, 23:845-851.
    • (2009) Leukemia , vol.23 , pp. 845-851
    • Lierman, E.1    Michaux, L.2    Beullens, E.3    Pierre, P.4    Marynen, P.5    Cools, J.6
  • 5
    • 46749115588 scopus 로고    scopus 로고
    • Ki11502, a novel multitargeted receptor tyrosine kinase inhibitor, induces growth arrest and apoptosis of human leukemia cells in vitro and in vivo
    • Nishioka C., Ikezoe T., Yang J., Miwa A., Tasaka T., Kuwayama Y., et al. Ki11502, a novel multitargeted receptor tyrosine kinase inhibitor, induces growth arrest and apoptosis of human leukemia cells in vitro and in vivo. Blood 2008, 111:5086-5092.
    • (2008) Blood , vol.111 , pp. 5086-5092
    • Nishioka, C.1    Ikezoe, T.2    Yang, J.3    Miwa, A.4    Tasaka, T.5    Kuwayama, Y.6
  • 6
    • 33747203008 scopus 로고    scopus 로고
    • Sorafenib is a potent inhibitor of FIP1L1-PDGFRalpha and the imatinib-resistant FIP1L1-PDGFRalpha T674I mutant
    • Lierman E., Folens C., Stover E.H., Mentens N., Van Miegroet H., Scheers W., et al. Sorafenib is a potent inhibitor of FIP1L1-PDGFRalpha and the imatinib-resistant FIP1L1-PDGFRalpha T674I mutant. Blood 2006, 108:1374-1376.
    • (2006) Blood , vol.108 , pp. 1374-1376
    • Lierman, E.1    Folens, C.2    Stover, E.H.3    Mentens, N.4    Van Miegroet, H.5    Scheers, W.6
  • 7
    • 33745059630 scopus 로고    scopus 로고
    • The FIP1L1-PDGFRA T674I mutation can be inhibited by the tyrosine kinase inhibitor AMN107 (nilotinib)
    • von Bubnoff N., Gorantla S.P., Thöne S., Peschel C., Duyster J. The FIP1L1-PDGFRA T674I mutation can be inhibited by the tyrosine kinase inhibitor AMN107 (nilotinib). Blood 2006, 107:4970-4971.
    • (2006) Blood , vol.107 , pp. 4970-4971
    • von Bubnoff, N.1    Gorantla, S.P.2    Thöne, S.3    Peschel, C.4    Duyster, J.5


* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.