Unilateral amyloid-β25-35 injection into the rat amygdala increases the expressions of aberrant tau phosphorylation kinases

Chinese Medical Journal

Volumn 123, Issue 10, 2010, Pages 1311-1314

  Huang, Jing ^a   Chen, Yu Juan ^a   Bian, Wei Hong ^a   Yu, Jing ^a   Zhao, Yu Wu ^b   Liu, Xue Yuan ^a  

^a TONGJI UNIVERSITY SCHOOL OF MEDICINE   (China)

^b SHANGHAI JIAO TONG UNIVERSITY AFFILIATED SIXTH PEOPLE S HOSPITAL   (China)

Author keywords

amyloid; Alzheimer disease; Glycogen synthase kinase 3 ; P38 mitogen activatedprotein kinase; Tau proteins

Indexed keywords

AMYLOID BETA PROTEIN[25-35]; GLYCOGEN SYNTHASE KINASE 3BETA; MESSENGER RNA; MITOGEN ACTIVATED PROTEIN KINASE P38; MONOCLONAL ANTIBODY; TAU PROTEIN; AMYLOID BETA PROTEIN; AMYLOID BETA PROTEIN (25 35); AMYLOID BETA-PROTEIN (25-35); GLYCOGEN SYNTHASE KINASE 3; GLYCOGEN SYNTHASE KINASE 3 BETA; PEPTIDE FRAGMENT; PHF 1; PHF-1;

ALZHEIMER DISEASE; AMYGDALOID NUCLEUS; ANIMAL TISSUE; ARTICLE; BRAIN CORTEX; CONTROLLED STUDY; DISEASE COURSE; DISEASE MODEL; FRONTAL CORTEX; HIPPOCAMPUS; IMMUNOHISTOCHEMISTRY; IN SITU HYBRIDIZATION; MALE; NONHUMAN; OPTICAL DENSITY; PAIRED HELICAL FILAMENT; PROTEIN EXPRESSION; PROTEIN PHOSPHORYLATION; RAT; ANIMAL; DRUG EFFECT; GENETICS; METABOLISM; PHOSPHORYLATION; SPRAGUE DAWLEY RAT;

AMYGDALA; AMYLOID BETA-PROTEIN; ANIMALS; ANTIBODIES, MONOCLONAL; GLYCOGEN SYNTHASE KINASE 3; IMMUNOHISTOCHEMISTRY; IN SITU HYBRIDIZATION; MALE; P38 MITOGEN-ACTIVATED PROTEIN KINASES; PEPTIDE FRAGMENTS; PHOSPHORYLATION; RATS; RATS, SPRAGUE-DAWLEY; TAU PROTEINS;

EID: 77952909384     PISSN: 03666999     EISSN: None     Source Type: Journal    
DOI: 10.3760/cma.j.issn.0366- 6999.2010.10.016     Document Type: Article

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