Abnormal mitochondrial function during ischemia reperfusion provides targets for pharmacological therapy.

Methodist DeBakey cardiovascular journal

Volumn 5, Issue 3, 2009, Pages 2-7

  García Rivas, Gerardo J ^a   Torre Amione, Guillermo ^a  

^a SCHOOL OF ENGINEERING AND SCIENCES   (Mexico)

Author keywords

[No Author keywords available]

Indexed keywords

ADENOSINE TRIPHOSPHATE; CALCIUM; CARDIOVASCULAR AGENT; CARRIER PROTEIN; MITOCHONDRIAL PERMEABILITY TRANSITION PORE; REACTIVE OXYGEN METABOLITE;

ANIMAL; APOPTOSIS; DRUG EFFECT; HEART ARRHYTHMIA; HEART MITOCHONDRION; HUMAN; METABOLISM; MITOCHONDRIAL MEMBRANE POTENTIAL; PATHOLOGY; REPERFUSION INJURY; REVIEW; SIGNAL TRANSDUCTION; TIME;

ADENOSINE TRIPHOSPHATE; ANIMALS; APOPTOSIS; ARRHYTHMIAS, CARDIAC; CALCIUM; CARDIOVASCULAR AGENTS; HUMANS; MEMBRANE POTENTIAL, MITOCHONDRIAL; MITOCHONDRIA, HEART; MITOCHONDRIAL MEMBRANE TRANSPORT PROTEINS; MYOCARDIAL REPERFUSION INJURY; REACTIVE OXYGEN SPECIES; SIGNAL TRANSDUCTION; TIME FACTORS;

EID: 77952766105     PISSN: 19476094     EISSN: None     Source Type: Journal    
DOI: 10.14797/mdcj-5-3-2     Document Type: Review

References (34)