Mitochondrial dysfunction and oxidative stress mediate the physiological impairment induced by the disruption of autophagy.

Aging

Volumn 1, Issue 4, 2009, Pages 425-437

  Wu, J Julie ^a   Quijano, Celia ^b   Chen, Edmund ^b   Liu, Hongjun ^b   Cao, Liu ^b   Fergusson, Maria M ^b   Rovira, Ilsa I ^b   Gutkind, Sarah ^b   Daniels, Mathew P ^b   Komatsu, Masaaki ^b   Finkel, Toren ^b  

^a NATIONAL HEART LUNG AND BLOOD INSTITUTE   (United States)

^b NONE

Author keywords

[No Author keywords available]

Indexed keywords

ATG7 PROTEIN, MOUSE; GLUCOSE; MICROTUBULE ASSOCIATED PROTEIN;

ANIMAL; ARTICLE; AUTOPHAGY; GENE EXPRESSION REGULATION; GENETICS; INSULIN RESISTANCE; MALE; METABOLISM; MITOCHONDRION; MOUSE; MOUSE MUTANT; OXIDATIVE STRESS; PANCREAS ISLET BETA CELL; PHYSIOLOGY;

ANIMALS; AUTOPHAGY; GENE EXPRESSION REGULATION; GLUCOSE; INSULIN RESISTANCE; INSULIN-SECRETING CELLS; MALE; MICE; MICE, KNOCKOUT; MICROTUBULE-ASSOCIATED PROTEINS; MITOCHONDRIA; OXIDATIVE STRESS;

MLCS; MLOWN;

EID: 77952409809     PISSN: None     EISSN: 19454589     Source Type: Journal    
DOI: 10.18632/aging.100038     Document Type: Article

References (0)