TCDD and a putative endogenous AhR Ligand, ITE, elicit the same immediate changes in gene expression in mouse lung fibroblasts

Toxicological Sciences

Volumn 114, Issue 1, 2009, Pages 90-100

  Henry, Ellen C ^a   Welle, Stephen L ^a   Gasiewicz, Thomas A ^a  

^a UNIVERSITY OF ROCHESTER MEDICAL CENTER   (United States)

Author keywords

Aryl hydrocarbon receptor (AhR); Endogenous ligand; Gene expression; Microarray; Mouse lung fibroblasts; TCDD

Indexed keywords

2 (1' H INDOLO 3' CARBONYL)THIAZOLE 4 CARBOXYLIC ACID METHYL ESTER; 2,3,7,8 TETRACHLORODIBENZO PARA DIOXIN; AROMATIC HYDROCARBON RECEPTOR; UNCLASSIFIED DRUG; XENOBIOTIC AGENT;

ANIMAL CELL; ARTICLE; CONFORMATIONAL TRANSITION; CONTROLLED STUDY; GENE EXPRESSION; LONG TERM EXPOSURE; LUNG FIBROBLAST; LUNG TOXICITY; MICROARRAY ANALYSIS; MOUSE; NONHUMAN; QUALITATIVE ANALYSIS; QUANTITATIVE ANALYSIS; REAL TIME POLYMERASE CHAIN REACTION; RESPIRATORY TRACT INFLAMMATION; TOXICOKINETICS; TRANSCRIPTION REGULATION;

MURINAE;

EID: 77951528539     PISSN: 10966080     EISSN: 10960929     Source Type: Journal    
DOI: 10.1093/toxsci/kfp285     Document Type: Article

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