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Volumn 12, Issue 3, 2010, Pages 185-193
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Aesculetin-induced apoptosis through a ROS-mediated mitochondrial dysfunction pathway in human cervical cancer cells
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Author keywords
Aesculetin; Apoptosis; HeLa cells; Mitochondrial
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Indexed keywords
CASPASE;
CYTOCHROME C;
ESCULETIN;
REACTIVE OXYGEN METABOLITE;
APOPTOSIS;
ARTICLE;
CANCER CELL;
CANCER CELL CULTURE;
CELL CYCLE G1 PHASE;
CELL FUNCTION;
CELL GROWTH;
CELL PROLIFERATION;
CELL STRUCTURE;
CELL VIABILITY;
CONTROLLED STUDY;
CYTOSOL;
CYTOTOXICITY;
DOSE RESPONSE;
DRUG EFFECT;
DRUG MECHANISM;
DRUG STRUCTURE;
ENZYME ACTIVITY;
GROWTH INHIBITION;
HELA CELL;
HUMAN;
HUMAN CELL;
IC 50;
MITOCHONDRION;
PROTEIN DEGRADATION;
UTERINE CERVIX CANCER;
ANTINEOPLASTIC AGENTS, PHYTOGENIC;
APOPTOSIS;
BISBENZIMIDAZOLE;
CASPASES;
CYTOCHROMES C;
DRUG SCREENING ASSAYS, ANTITUMOR;
FEMALE;
HELA CELLS;
HUMANS;
MITOCHONDRIA;
MODELS, BIOLOGICAL;
MOLECULAR STRUCTURE;
PLANTS, MEDICINAL;
TUMOR CELLS, CULTURED;
UMBELLIFERONES;
UTERINE CERVICAL NEOPLASMS;
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EID: 77951474131
PISSN: 10286020
EISSN: 14772213
Source Type: Journal
DOI: 10.1080/10286020903427336 Document Type: Article |
Times cited : (46)
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References (25)
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