Variant Ph translocations in chronic myeloid leukemia
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Mechanisms of genesis of variant translocation in chronic myeloid leukemia are not correlated with ABL1 or BCR deletion status or response to imatinib therapy
Richebourg S, Eclache V, Perot C, Portnoi MF, Van den Akker J, Terré C, et al. Mechanisms of genesis of variant translocation in chronic myeloid leukemia are not correlated with ABL1 or BCR deletion status or response to imatinib therapy. Cancer Genet Cytogenet 2008;182:95-102.
Evolving concepts in the management of chronic myeloid leukemia: Recommendations from an expert panel on behalf of the European LeukemiaNet
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Monitoring CML patients responding to treatment with tyrosine kinase inhibitors: Review and recommendations for harmonizing current methodology for detecting BCR-ABL transcripts and kinase domain mutations and for expressing results
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Does a rise in the BCR-ABL1 transcript level identify chronic phase CML patients responding to imatinib who have a high risk of cytogenetic relapse?
Marin D, Khorashad JS, Foroni L, Milojkovic D, Szydlo R, Reid AG, et al. Does a rise in the BCR-ABL1 transcript level identify chronic phase CML patients responding to imatinib who have a high risk of cytogenetic relapse? Br J Haematol 2009;145:373-375
On the genesis and prognosis of variant translocations in chronic myeloid leukemia
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Results of imatinib mesylate therapy in chronic myelogenous leukaemia with variant Philadelphia chromosome
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