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Volumn 121, Issue 10, 2010, Pages 1244-1255
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Thioredoxin-1 gene therapy enhances angiogenic signaling and reduces ventricular remodeling in infarcted myocardium of diabetic rats.
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Author keywords
[No Author keywords available]
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Indexed keywords
HEME OXYGENASE 1;
MITOGEN ACTIVATED PROTEIN KINASE P38;
STREPTOZOCIN;
STRESS ACTIVATED PROTEIN KINASE;
THIOREDOXIN;
VASCULOTROPIN A;
ANGIOGENESIS;
ANIMAL;
APOPTOSIS;
ARTICLE;
CELL CULTURE;
ENDOTHELIUM CELL;
EXPERIMENTAL DIABETES MELLITUS;
FIBROSIS;
GENE THERAPY;
GENETIC TRANSFECTION;
GENETICS;
HEART INFARCTION;
HEART MUSCLE;
HEART VENTRICLE REMODELING;
HUMAN;
MALE;
METABOLISM;
OXIDATIVE STRESS;
PATHOLOGY;
PATHOPHYSIOLOGY;
RAT;
SIGNAL TRANSDUCTION;
SPRAGUE DAWLEY RAT;
ANIMALS;
APOPTOSIS;
CELLS, CULTURED;
DIABETES MELLITUS, EXPERIMENTAL;
ENDOTHELIAL CELLS;
FIBROSIS;
GENE THERAPY;
HEME OXYGENASE-1;
HUMANS;
JNK MITOGEN-ACTIVATED PROTEIN KINASES;
MALE;
MYOCARDIAL INFARCTION;
MYOCARDIUM;
NEOVASCULARIZATION, PHYSIOLOGIC;
OXIDATIVE STRESS;
P38 MITOGEN-ACTIVATED PROTEIN KINASES;
RATS;
RATS, SPRAGUE-DAWLEY;
SIGNAL TRANSDUCTION;
STREPTOZOCIN;
THIOREDOXINS;
TRANSFECTION;
VASCULAR ENDOTHELIAL GROWTH FACTOR A;
VENTRICULAR REMODELING;
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EID: 77950496435
PISSN: None
EISSN: 15244539
Source Type: Journal
DOI: 10.1161/CIRCULATIONAHA.109.872481 Document Type: Article |
Times cited : (104)
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References (0)
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