Autophagy in nuclear receptor PPARγ-deficient mouse prostatic carcinogenesis

Autophagy

Volumn 6, Issue 1, 2010, Pages 175-176

  Jiang, Ming ^a   Jerome, W Gray ^a   Hayward, Simon W ^a  

^a VANDERBILT INGRAM CANCER CENTER   (United States)

Author keywords

Autophagy; Differentiation; Fatty acid oxidation; Oxidative stress; Peroxisome proliferator activated receptor (PPAR) ; PIN; Prostatic intraepithelial neoplasia

Indexed keywords

CELL NUCLEUS RECEPTOR; PEROXISOME PROLIFERATOR ACTIVATED RECEPTOR GAMMA; PEROXISOME PROLIFERATOR ACTIVATED RECEPTOR GAMMA 2;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; AUTOPHAGY; CARCINOGENESIS; CELL CYCLE REGULATION; CELL DEDIFFERENTIATION; CELL PROLIFERATION; CELL VACUOLE; ELECTRON MICROSCOPY; FATTY ACID OXIDATION; GENE EXPRESSION PROFILING; HISTOPATHOLOGY; IN VITRO STUDY; IN VIVO STUDY; LIPID DEGRADATION; LIPID OXIDATION; LYSOSOME; METABOLISM; MITOCHONDRION; MOUSE; NONHUMAN; NULL CELL; OXIDATIVE STRESS; PEROXISOME; PHENOTYPE; PROSTATE EPITHELIUM; PROSTATIC INTRAEPITHELIAL NEOPLASIA; SIGNAL TRANSDUCTION;

MURINAE;

EID: 75149140978     PISSN: 15548627     EISSN: 15548635     Source Type: Journal    
DOI: 10.4161/auto.6.1.10700     Document Type: Article

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