Why do cone photoreceptors die in rod-specific forms of retinal degenerations

Ophthalmic Genetics

Volumn 30, Issue 3, 2009, Pages 152-154

  Koenekoop, Robert K ^a  

^a MCGILL UNIVERSITY HEALTH CENTRE   (Canada)

Author keywords

Blindness; Cone death; Insulin mTOR; Retinitis pigmentosa; Rod death

Indexed keywords

GLUCOSE TRANSPORTER 1; HYPOXIA INDUCIBLE FACTOR 1; LYSOSOME ASSOCIATED MEMBRANE PROTEIN 2; MAMMALIAN TARGET OF RAPAMYCIN;

APOPTOSIS; AUTOPHAGY; GENE MUTATION; IMMUNOHISTOCHEMISTRY; MUELLER CELL; NICK END LABELING; NIGHT BLINDNESS; NONHUMAN; PATHOGENESIS; PRIORITY JOURNAL; REAL TIME POLYMERASE CHAIN REACTION; RETINA CONE; RETINA ROD; RETINITIS PIGMENTOSA; SHORT SURVEY; VISUAL ACUITY; VISUAL FIELD DEFECT;

ANIMALS; APOPTOSIS; AUTOPHAGY; HUMANS; RETINAL CONE PHOTORECEPTOR CELLS; RETINAL ROD PHOTORECEPTOR CELLS; RETINITIS PIGMENTOSA;

ANIMALIA;

EID: 70350635813     PISSN: 13816810     EISSN: 17445094     Source Type: Journal    
DOI: 10.1080/13816810902788974     Document Type: Short Survey

References (1)

1. Punzo C,Kornacker K, Cepko CL. Stimulation of the insulin/mTOR pathway delays cone death in a mouse model of retinitis pigmentosa. Nat Neurosci. 2008;12:44-52.