Function of Ca(2+) release channels in Purkinje cells that survive in the infarcted canine heart: a mechanism for triggered Purkinje ectopy.

Circulation. Arrhythmia and electrophysiology

Volumn 1, Issue 5, 2008, Pages 387-395

  Hirose, Masanori ^a   Stuyvers, Bruno D ^a   Dun, Wen ^a   ter Keurs, Henk E D J ^a   Boyden, Penelope A ^a  

^a Och Spine at New York Presbyterian Hospitals

Author keywords

[No Author keywords available]

Indexed keywords

CAFFEINE; CALCIUM CHANNEL; K201 COMPOUND; SAPONIN; TETRACAINE; THIAZEPINE DERIVATIVE;

ANIMAL; ARTICLE; CALCIUM SIGNALING; CELL SURVIVAL; CONFOCAL MICROSCOPY; DISEASE MODEL; DOG; DOSE RESPONSE; DRUG EFFECT; HEART ARRHYTHMIA; HEART INFARCTION; KINETICS; METABOLISM; PATHOLOGY; PATHOPHYSIOLOGY; PERMEABILITY; PURKINJE CELL; SARCOPLASMIC RETICULUM;

ANIMALS; ARRHYTHMIAS, CARDIAC; CAFFEINE; CALCIUM CHANNELS; CALCIUM SIGNALING; CELL SURVIVAL; DISEASE MODELS, ANIMAL; DOGS; DOSE-RESPONSE RELATIONSHIP, DRUG; KINETICS; MICROSCOPY, CONFOCAL; MYOCARDIAL INFARCTION; PERMEABILITY; PURKINJE CELLS; SAPONINS; SARCOPLASMIC RETICULUM; TETRACAINE; THIAZEPINES;

EID: 70350435971     PISSN: None     EISSN: 19413084     Source Type: Journal    
DOI: 10.1161/CIRCEP.107.758110     Document Type: Article

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