Redox imbalance provokes deactivation of macrophages in sepsis

Proteomics - Clinical Applications

Volumn 3, Issue 8, 2009, Pages 1000-1009

  Chen, Ting ^a   Lin, Xuzhu ^a   Xu, Jiafa ^a   Tan, Renxiang ^a   Ji, Jianguo ^b   Shen, Pingping ^a  

^a NANJING UNIVERSITY   (China)

^b PEKING UNIVERSITY   (China)

Author keywords

Macrophages; Oxidation; Reduction; ROS; Sepsis

Indexed keywords

GLUTATHIONE; HYDROGEN PEROXIDE; I KAPPA B; IMMUNOGLOBULIN ENHANCER BINDING PROTEIN; LIPOPOLYSACCHARIDE; MITOGEN ACTIVATED PROTEIN KINASE; REACTIVE OXYGEN METABOLITE; TOLL LIKE RECEPTOR; TRANSCRIPTION FACTOR AP 1;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; BINDING AFFINITY; CELL DEATH; CONTROLLED STUDY; CYTOKINE RELEASE; DNA BINDING; ENZYME PHOSPHORYLATION; GEL MOBILITY SHIFT ASSAY; IMMUNE DEFICIENCY; IMMUNOSUPPRESSIVE TREATMENT; INFLAMMATORY CELL; INNATE IMMUNITY; MACROPHAGE ACTIVATION; MALE; MATRIX ASSISTED LASER DESORPTION IONIZATION TIME OF FLIGHT MASS SPECTROMETRY; MOUSE; NONHUMAN; OXIDATION REDUCTION STATE; OXIDATIVE STRESS; PATHOGENESIS; PHAGOCYTOSIS; PHENOTYPE; PRIORITY JOURNAL; PROTEIN EXPRESSION; SEPSIS; TWO DIMENSIONAL ELECTROPHORESIS; UPREGULATION; WESTERN BLOTTING;

EID: 70350223567     PISSN: 18628346     EISSN: None     Source Type: Journal    
DOI: 10.1002/prca.200800016     Document Type: Article

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