Glomerular disease: The Oxford classificationpredicting progression of IgAN

Nature Reviews Nephrology

Volumn 5, Issue 10, 2009, Pages 557-559

  Eitner, Frank ^a   Floege, Jürgen ^a  

^a RWTH AACHEN UNIVERSITY   (Germany)

Author keywords

[No Author keywords available]

Indexed keywords

DISEASE CLASSIFICATION; DISEASE COURSE; GLOMERULOPATHY; HUMAN; IMMUNOGLOBULIN A NEPHROPATHY; KIDNEY BIOPSY; LUPUS ERYTHEMATOSUS NEPHRITIS; MEDICAL DECISION MAKING; PRIORITY JOURNAL; SHORT SURVEY;

DISEASE PROGRESSION; GLOMERULONEPHRITIS, IGA; HUMANS; KIDNEY GLOMERULUS; PROGNOSIS;

EID: 70349618909     PISSN: 17595061     EISSN: 1759507X     Source Type: Journal    
DOI: 10.1038/nrneph.2009.150     Document Type: Short Survey

References (6)

1. Roberts, i. s. et al. The Oxford classification of igA nephropathy: pathology definitions, correlations, and reproducibility. Kidney Int. 76, 546-556 (2009).
2. Cattran, D. C. et al. The Oxford classification of igA nephropathy: rationale, clinicopathological correlations, and classification. Kidney Int. 76, 534-545 (2009).
3. weening, J. J. et al. The classification of glomerulonephritis in systemic lupus erythematosus revisited. Kidney Int. 65, 521-530 (2004).
4. eitner, F., Ackermann, D., Hilgers, R. D. & Floege, J. supportive versus immunosuppressive therapy of progressive igA nephropathy (sTOP) igAn trial: rationale and study protocol. J. Nephrol. 21, 284-289 (2008).
5. suzuki, H. et al. Aberrantly glycosylated igA1 in igA nephropathy patients is recognized by igG antibodies with restricted heterogeneity. J. Clin. Invest. 119, 1668-1677 (2009).
6. Gharavi, A. G. et al. igA nephropathy, the most common cause of glomerulonephritis, is linked to 6q22-23. Nat. Genet. 26, 354-357 (2000).