Bile acids as endogenous etiologic agents in gastrointestinal cancer

World Journal of Gastroenterology

Volumn 15, Issue 27, 2009, Pages 3329-3340

  Bernstein, Harris ^a   Bernstein, Carol ^a,b   Payne, Claire M ^a   Dvorak, Katerina ^a,b  

^a UNIVERSITY OF ARIZONA COLLEGE OF MEDICINE   (United States)

^b SOUTHERN ARIZONA VA HEALTH CARE SYSTEM   (United States)

Author keywords

Adenocarcinoma; Apoptosis; Bile acids; Cancer; Colon; DNA damage; Esophagus; Pancreas; Small intestine; Stomach

Indexed keywords

2 [[4 [2,6 BIS(1 PYRROLIDINYL) 4 PYRIMIDINYL] 1 PIPERAZINYL]METHYL] 3,4 DIHYDRO 2,5,7,8 TETRAMETHYL 2H 1 BENZOPYRAN 6 OL; ALPHA TOCOPHEROL; CYCLOOXYGENASE 2; DEOXYCHOLIC ACID; EBSELEN; FARNESOID X RECEPTOR; GLYCOCHENODEOXYCHOLIC ACID; GLYCOCHOLIC ACID; GLYCODEOXYCHOLIC ACID; PREGNANE X RECEPTOR; REACTIVE NITROGEN SPECIES; REACTIVE OXYGEN METABOLITE; TAUROCHOLIC ACID; VITAMIN D RECEPTOR; BILE ACID; CARCINOGEN; CELL RECEPTOR;

APOPTOSIS; BILIARY TRACT CANCER; CANCER RISK; CARCINOGENESIS; COLON CANCER; DIETARY INTAKE; DIGESTIVE SYSTEM CANCER; DNA DAMAGE; DRUG MECHANISM; EDITORIAL; ESOPHAGUS CANCER; GENE MUTATION; HELICOBACTER INFECTION; HEPATITIS; HUMAN; LIPID ANALYSIS; LIVER CANCER; NONHUMAN; OXIDATIVE STRESS; PANCREAS CANCER; RECTUM CANCER; SEX DIFFERENCE; SMALL INTESTINE CANCER; STOMACH CANCER; STOMACH PH; ANIMAL; CELL TRANSFORMATION; FEMALE; GASTROINTESTINAL TUMOR; MALE; METABOLISM; PATHOLOGY; PATHOPHYSIOLOGY; PHYSIOLOGY; RISK FACTOR;

ANIMALS; APOPTOSIS; BILE ACIDS AND SALTS; CARCINOGENS; CELL TRANSFORMATION, NEOPLASTIC; DNA DAMAGE; FEMALE; GASTROINTESTINAL NEOPLASMS; HUMANS; MALE; REACTIVE OXYGEN SPECIES; RECEPTORS, CYTOPLASMIC AND NUCLEAR; RISK FACTORS;

EID: 70349563339     PISSN: 10079327     EISSN: None     Source Type: Journal    
DOI: 10.3748/wjg.15.3329     Document Type: Editorial

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