Host shutoff is a conserved phenotype of gammaherpesvirus infection and is orchestrated exclusively from the cytoplasm

Journal of Virology

Volumn 83, Issue 18, 2009, Pages 9554-9566

  Covarrubias, Sergio ^a   Richner, Justin M ^b   Clyde, Karen ^b   Lee, Yeon J ^b   Glaunsinger, Britt A ^b  

^a UNIVERSITY OF CALIFORNIA   (United States)

^b UNIVERSITY OF CALIFORNIA   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

MESSENGER RNA; POLYADENYLIC ACID BINDING PROTEIN; VIRUS PROTEIN;

ADENYLATION; ANIMAL CELL; ARTICLE; CELL NUCLEUS; CONTROLLED STUDY; CYTOPLASM; EMBRYO; EXPERIMENTAL MODEL; GAMMAHERPESVIRUS; GENE CONTROL; GENE EXPRESSION REGULATION; GENE FUNCTION; GENE TARGETING; HERPES; HERPES VIRUS; HUMAN; HUMAN CELL; IN VIVO STUDY; MOUSE; NONHUMAN; PHENOTYPE; PRIORITY JOURNAL; PROTEIN FUNCTION; PROTEIN LOCALIZATION; SOWING DATE; TARGET CELL; VIRUS GENE; VIRUS PATHOGENESIS; VIRUS STRAIN;

ANIMALS; CYTOPLASM; DEOXYRIBONUCLEASES; GAMMAHERPESVIRINAE; HERPESVIRIDAE INFECTIONS; HERPESVIRUS 4, HUMAN; HERPESVIRUS 8, HUMAN; HOST-PATHOGEN INTERACTIONS; HUMANS; MICE; RHADINOVIRUS; RNA, MESSENGER; TUMOR VIRUS INFECTIONS; VIRAL PROTEINS;

HUMAN HERPESVIRUS 4; HUMAN HERPESVIRUS 8; MURINAE;

EID: 69449095602     PISSN: 0022538X     EISSN: None     Source Type: Journal    
DOI: 10.1128/JVI.01051-09     Document Type: Article

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