Comment on: Excessive opioid use and the development of chronic migraine

Pain

Volumn 145, Issue 1-2, 2009, Pages 262-263

  Bartley, Jim ^a   Watkins, Linda R ^b  

^a The Auckland Regional Pain Service   (New Zealand)

^b UNIVERSITY OF COLORADO   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

MORPHINE; OPIATE;

ALLODYNIA; ANALGESIA; CELL ACTIVATION; DRUG TOLERANCE; GLIA CELL; HYPERALGESIA; LETTER; NERVE CELL; PRIORITY JOURNAL; TRANSFORMED MIGRAINE;

ANALGESICS, OPIOID; HUMANS; MIGRAINE DISORDERS;

EID: 69249208460     PISSN: 03043959     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.pain.2009.06.007     Document Type: Letter

References (4)

1. Bartley J. Could glial activation be a factor in migraine?. Med Hypotheses 72 (2009) 255-257
2. Bigal M.E., and Lipton R.B. Excessive opioid use and the development of chronic migraine. Pain 142 (2009) 179-182
3. Hutchinson M.R., Bland S.T., Johnson K.W., Rice K.C., Maier S.F., and Watkins L.R. Opioid-induced glial activation: mechanisms of activation and implications for opioid analgesia, dependence, and reward. ScientificWorldJ 7 (2007) 98-111
4. Shavit Y., Wolf G., Johnston I.N., Westbrook R.F., Watkins L.R., and Yirmiya R. Proinflammatory cytokines modulate neuropathic pain, opioid analgesia and opioid tolerance. In: De Leo J.A., Sorkin L.A., and Watkins L.R. (Eds). Immune and glial regulation of pain (2007), IASP Press, Seattle 361-382