Response to exercise generates lactate and fluid intake: Effects on mitochondrial function in heart and vascular smooth muscle

Hypertension

Volumn 54, Issue 2, 2009, Pages

  Miyachi, Masaaki ^a   Nagata, Kohzo ^b  

^a NAGOYA UNIVERSITY GRADUATE SCHOOL OF MEDICINE   (Japan)

^b NAGOYA UNIVERSITY   (Japan)

Author keywords

[No Author keywords available]

Indexed keywords

LACTIC ACID; MAMMALIAN TARGET OF RAPAMYCIN; MONOCARBOXYLATE TRANSPORTER 1; PEROXISOME PROLIFERATOR ACTIVATED RECEPTOR GAMMA COACTIVATOR 1ALPHA; PROTEIN KINASE B;

ANGIOGENESIS; EXERCISE; FLUID INTAKE; HEART MITOCHONDRION; HUMAN; HYPOXIA; LETTER; MITOCHONDRIAL RESPIRATION; MUSCLE MITOCHONDRION; NONHUMAN; PRIORITY JOURNAL; SIGNAL TRANSDUCTION; VASCULAR SMOOTH MUSCLE;

EID: 68749102198     PISSN: 0194911X     EISSN: None     Source Type: Journal    
DOI: 10.1161/HYPERTENSIONAHA.109.135285     Document Type: Letter

References (5)

1. Thornton SN, Hess K. Exercise generates lactate and fluid intake: effects on mitochondrial function in heart and vascular smooth muscle. Hypertension. 2009;54:e14.
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3. Schulze PC. Beneficial effects of moderate exercise in arterial hypertension. Hypertension. 2009;53:600-601.
4. Cunningham JT, Rodgers JT, Arlow DH, Vazquez F, Mootha VK, Puigserver P. mTOR controls mitochondrial oxidative function through a YY1-PGC-1′ transcriptional complex. Nature. 2007;450:736-740.
5. Schliess F, Richter L, vom Dahl S, Häussinger D. Cell hydration and mTOR-dependent signalling. Acta Physiol (Oxf). 2006;187:223-229.