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Volumn 26, Issue 5, 2009, Pages 781-788
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Disruption of the ubiquitin proteasome system following axonal stretch injury accelerates progression to secondary axotomy.
a
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Author keywords
[No Author keywords available]
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Indexed keywords
ACETYLCYSTEINE;
BENZYLOXYCARBONYLLEUCYL LEUCYL LEUCINE ALDEHYDE;
BENZYLOXYCARBONYLLEUCYL-LEUCYL-LEUCINE ALDEHYDE;
CYSTEINE PROTEINASE INHIBITOR;
DRUG DERIVATIVE;
LACTACYSTIN;
LEUPEPTIN;
NEUROFILAMENT PROTEIN;
PROTEASOME;
UBIQUITIN;
ANIMAL;
ARTICLE;
CELL CULTURE;
DRUG ANTAGONISM;
DRUG EFFECT;
IMMUNOHISTOCHEMISTRY;
METABOLISM;
NERVE CELL;
NERVE FIBER;
NERVE FIBER TRANSECTION;
PATHOLOGY;
PHOSPHORYLATION;
PHYSIOLOGY;
RAT;
STIMULATION;
UBIQUITINATION;
WISTAR RAT;
ACETYLCYSTEINE;
ANIMALS;
AXONS;
AXOTOMY;
CELLS, CULTURED;
CYSTEINE PROTEINASE INHIBITORS;
IMMUNOHISTOCHEMISTRY;
LEUPEPTINS;
NEUROFILAMENT PROTEINS;
NEURONS;
PHOSPHORYLATION;
PHYSICAL STIMULATION;
PROTEASOME ENDOPEPTIDASE COMPLEX;
RATS;
RATS, WISTAR;
UBIQUITIN;
UBIQUITINATION;
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EID: 68349148802
PISSN: None
EISSN: 15579042
Source Type: Journal
DOI: 10.1089/neu.2008.0669 Document Type: Article |
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Times cited : (20)
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References (0)
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