WRN helicase defective in the premature aging disorder Werner syndrome genetically interacts with topoisomerase 3 and restores the top3 slow growth phenotype of sgs1 top3.

Aging

Volumn 1, Issue 2, 2009, Pages 219-233

  Aggarwal, Monika ^a   Brosh, Robert M ^b  

^a NATIONAL INSTITUTES OF HEALTH   (United States)

^b NONE

Author keywords

[No Author keywords available]

Indexed keywords

DNA TOPOISOMERASE; DNA TOPOISOMERASE III; EXODEOXYRIBONUCLEASE; RECQ HELICASE; SACCHAROMYCES CEREVISIAE PROTEIN; SGS1 PROTEIN, S CEREVISIAE; WRN PROTEIN, HUMAN;

ARTICLE; ENZYMOLOGY; GENE EXPRESSION REGULATION; GENETIC POLYMORPHISM; GENETIC RECOMBINATION; GENETICS; HUMAN; METABOLISM; MISSENSE MUTATION; PHYSIOLOGY; SACCHAROMYCES CEREVISIAE; WERNER SYNDROME;

DNA TOPOISOMERASES, TYPE I; EXODEOXYRIBONUCLEASES; GENE EXPRESSION REGULATION, FUNGAL; HUMANS; MUTATION, MISSENSE; POLYMORPHISM, GENETIC; RECOMBINATION, GENETIC; RECQ HELICASES; SACCHAROMYCES CEREVISIAE; SACCHAROMYCES CEREVISIAE PROTEINS; WERNER SYNDROME;

MLCS; MLOWN;

EID: 68049113833     PISSN: None     EISSN: 19454589     Source Type: Journal    
DOI: 10.18632/aging.100020     Document Type: Article

References (0)