Goodpasture syndrome associated with p-ANCA microscopic vasculitis: A rare entity to be know;Syndrome de Goodpasture associé à une vasculite microscopique à p-ANCA: Une entité rare à ne pas méconnaî tre

Revue Medicale Suisse

Volumn 5, Issue 207, 2009, Pages 1330-1334

  Pralong, Géraldine ^a   Fournier, Christian ^a   Meier, Pascal ^a   Dayer, Eric ^b  

^a Hopital de Sion   (Switzerland)

^b Institut Central des Hopitaux Valaisans   (Switzerland)

Author keywords

[No Author keywords available]

Indexed keywords

AZATHIOPRINE; BASEMENT MEMBRANE ANTIBODY; CORTICOSTEROID; CYCLOPHOSPHAMIDE; MYCOPHENOLIC ACID 2 MORPHOLINOETHYL ESTER;

ADULT; ANCA ASSOCIATED VASCULITIS; AUTOIMMUNE DISEASE; BASEMENT MEMBRANE; CASE REPORT; CLINICAL FEATURE; GLOMERULONEPHRITIS; GOODPASTURE SYNDROME; HISTOPATHOLOGY; HUMAN; HUMAN TISSUE; IMMUNOFLUORESCENCE; IMMUNOSUPPRESSIVE TREATMENT; NEEDLE BIOPSY; PATHOPHYSIOLOGY; PROGNOSIS; REVIEW;

ANTI-GLOMERULAR BASEMENT MEMBRANE DISEASE; ANTIBODIES, ANTINEUTROPHIL CYTOPLASMIC; AUTOANTIBODIES; BIOLOGICAL MARKERS; DIABETES COMPLICATIONS; HUMANS; IMMUNOLOGIC FACTORS; IMMUNOSUPPRESSIVE AGENTS; MALE; MIDDLE AGED; PROGNOSIS; TREATMENT OUTCOME; VASCULITIS;

EID: 67649845732     PISSN: 16609379     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Review

References (15)

1. ** Lionaki S, Jennette JC, Falk RJ. Anti-neutrophil cytoplasmic (ANCA) and anti-glomerular basement membrane (GBM) autoantibodies in necrotizing and crescentic glomerulonephritis. Semin Immunopathol 2007;29:459-74.
2. * Pusey CD. Anti-glomerular basement membrane disease. Kidney Int 2003;64:1535-50.
3. * Guilpain P, Servettaz A, Batteux F, Guillevin L, Mouthon L. Natural and disease associated anti-myeloperoxidase (MPO) autoantibodies. Autoimmun Rev 2008;6:421-5.
4. * Rutgers A, Slot M, van Paassen P, et al. Coexistence of anti-glomerular basement membrane antibodies and myeloperoxidase-ANCAs in crescentic glomerulonephritis. Am J Kidney Dis 2005;46:253-62.
5. * Levy JB, Hammad T, Coulthart A, Dougan T, Pusey CD. Clinical features and outcome of patients with both ANCA and anti-GBM antibodies. Kidney Int 2004;66:1535-40.
6. Zhao J, Yang R, Cui Z, et al. Characteristics and outcome of Chinese patients with both antineutrophil cytoplasmic antibody and antiglomerular basement membrane antibodies. Nephron Clin Pract 2007;107:c56-62.
7. Kinjoh K, Kyogoku M, Good RA. Genetic selection for crescent formation yields mouse strain with rapidly progressive glomerulonephritis and small vessel vasculitis. Proc Natl Acad Sci USA 1993;90:3413-7.
8. Xiao H, Heeringa P, Hu P, et al. Antineutrophil cytoplasmic autoantibodies specific for myeloperoxidase cause glomerulonephritis and vasculitis in mice. J Clin Invest 2002;110:955-63.
9. Chan TM, Frampton G, Jayne DR, et al. Clinical significance of anti-endothelial cell antibodies in systemic vasculitis: A longitudinal study comparing anti-endothelial cell antibodies and anti-neutrophil cytoplasm antibodies. Am J Kidney Dis 1993;22:387-92.
10. * Kain R, Exner M, Brandes R, et al. Molecular mimicry in pauci-immune focal necrotizing glomerulonephritis. Nat Med 2008;14:1088-96.
11. Levy JB, Turner AN, Rees AJ, Pusey CD. Long-term outcome of anti-glomerular basement membrane antibody disease treated with plasma exchange and immunosuppression. Ann Intern Med 2001;134:1033-42.
12. * Segelmark M, Hellmark T, Wieslander J. The prognostic significance in Goodpasture's disease of specificity, titre and affinity of anti-glomerular-basement-membrane antibodies. Nephron Clin Pract 2003;94:c59-68.
13. * Slot MC, Tervaert JW, Franssen CF, Stegeman CA. Renal survival and prognostic factors in patients with PR3-ANCA associated vasculitis with renal involvement. Kidney Int 2003;63:670-7.
14. Bosch X, Mirapeix E, Font J, et al. Prognostic implication of anti-neutrophil cytoplasmic autoantibodies with myeloperoxidase specificity in anti-glomerular basement membrane disease. Clin Nephrol 1991;36:107-13.
15. Ferraro AJ, Smith SW, Neil D, Savage CO. Relapsed Wegener's granulomatosis after rituximab therapy - B cells are present in new pathological lesions despite persistent «depletion» of peripheral blood. Nephrol Dial Transplant 2008;23:3030-2.