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Volumn 485, Issue 1, 2009, Pages 82-87

Roles of ERK, PI3 kinase, and PLC-γ pathways induced by overexpression of translationally controlled tumor protein in HeLa cells

(3) Kim, Moonhee a Jung, Jaehoon a Lee, Kyunglim a

a Ewha Womans University (South Korea)

Author keywords

EGFR; ERK; Na,K ATPase; Ouabain; PI3K; PLC ; TCTP

Indexed keywords

ADENOSINE TRIPHOSPHATASE (POTASSIUM SODIUM); ADENOVIRUS VECTOR; EPIDERMAL GROWTH FACTOR RECEPTOR; MITOGEN ACTIVATED PROTEIN KINASE; PHOSPHATIDYLINOSITOL 3 KINASE; PHOSPHOLIPASE C GAMMA; PROTEIN KINASE B; TUMOR PROTEIN;

APOPTOSIS; ARTICLE; CELL MIGRATION; CELL SURVIVAL; ENZYME INHIBITION; HELA CELL; HUMAN; HUMAN CELL; HUMAN CELL CULTURE; IMMUNOBLOTTING; IMMUNOPRECIPITATION; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN PHOSPHORYLATION; PROTEIN PROCESSING; SIGNAL TRANSDUCTION;

1-PHOSPHATIDYLINOSITOL 3-KINASE; ADENOVIRIDAE; ANIMALS; EXTRACELLULAR SIGNAL-REGULATED MAP KINASES; GENE EXPRESSION; HELA CELLS; HUMANS; PHOSPHOLIPASE C GAMMA; PHOSPHORYLATION; PROTO-ONCOGENE PROTEINS C-AKT; PROTO-ONCOGENE PROTEINS C-RAF; RAS PROTEINS; RECEPTOR, EPIDERMAL GROWTH FACTOR; SIGNAL TRANSDUCTION; SODIUM-POTASSIUM-EXCHANGING ATPASE; TUMOR MARKERS, BIOLOGICAL;

ADENOVIRIDAE; STROPHANTHUS GRATUS;

EID: 64549105686 PISSN: 00039861 EISSN: 10960384 Source Type: Journal
DOI: 10.1016/j.abb.2009.02.002 Document Type: Article

Times cited : (27)

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