Impairment in reactivation of a latency associated transcript (LAT)-deficient HSV-2 is not solely dependent on the latent viral load or the number of CD8+ T cells infiltrating the ganglia

Virology

Volumn 387, Issue 1, 2009, Pages 193-199

  Hoshino, Yo ^a   Pesnicak, Lesley ^a   Straus, Stephen E ^a   Cohen, Jeffrey I ^a  

^a NATIONAL INSTITUTE OF ALLERGY AND INFECTIOUS DISEASES   (United States)

Author keywords

CD8+ T cells; Herpes simplex virus; Latency; Latency associated transcripts; Reactivation; Viral load; Wide range inocula

Indexed keywords

LATENCY ASSOCIATED PEPTIDE;

ANIMAL CELL; ANIMAL MODEL; ARTICLE; CD8+ T LYMPHOCYTE; CELL COUNT; CELL INFILTRATION; COMPARATIVE STUDY; EX VIVO STUDY; GANGLION; HERPES SIMPLEX VIRUS 2; INOCULATION; MOUSE; NONHUMAN; PRIORITY JOURNAL; VIRUS CULTURE; VIRUS DETECTION; VIRUS LATENCY; VIRUS LOAD; VIRUS REACTIVATION; VIRUS TRANSCRIPTION;

ANIMALS; CD8-POSITIVE T-LYMPHOCYTES; CELLS, CULTURED; GENE EXPRESSION REGULATION, VIRAL; HERPES SIMPLEX; HERPESVIRUS 2, HUMAN; MICE; TRIGEMINAL GANGLION; VIRAL LOAD; VIRAL PROTEINS; VIRUS ACTIVATION; VIRUS LATENCY;

HUMAN HERPESVIRUS 2; MUS; SIMPLEXVIRUS;

EID: 63549107828     PISSN: 00426822     EISSN: 10960341     Source Type: Journal    
DOI: 10.1016/j.virol.2009.02.004     Document Type: Article

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