Correlation between the intramucosal growth type and the incidence of P53-overexpression and point mutations of K-RAS gene in the development of colorectal cancer less than 20MM in diameter

Gastroenterological Endoscopy

Volumn 39, Issue 4, 1997, Pages 784-785

  Otsuka, Motoko ^a   Nagasako, Kou ^b   Sano, Yasushi ^c   Fujimori, Takahiro ^d   Hayashi, Naoaki ^a  

^a TOKYO WOMEN'S MEDICAL UNIVERSITY   (Japan)

^b Gunma Cancer Institute   (Japan)

^c DOKKYO MEDICAL UNIVERSITY   (Japan)

^d NONE

Author keywords

[No Author keywords available]

Indexed keywords

EID: 6244303681     PISSN: 03871207     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

References (12)

1. Ikegami M. A pathological study on colorectal cancer. Acta Pathol Jpn 1987 ; 37 : 21-37.
2. Feig LA. The many road that lead to ras. Science 1993 ; 260 : 767-8.
3. Chung H H, Benson D R, Schultz P G. Probing the structure and mechanism of ras protein with an expanded genetic code. Science 1993 ; 259 : 806-9.
4. Yukawa M, Fujimori T, Maeda S et al. Comparalive clinicopathological and immunohistochemical study of ras and P53 in flat and polypoid type colorecctal tumours. Gut 1994 ; 35 : 1258-61.
5. Yamagata S, Muto T, Uchida Y et al. Lower incidence of K-ras codon 12 mutation in flat colorectal adenomas than in polypoid adenomas. Jpn. J. Cancer Res 1994 ; 85 : 147-51.
6. Fujimori T, Satonaka K, Idei Y et al. Non-involvement of ras mutations in flat colorectal adenomas and carcinomas. Int. J Cancer 1994 ; 57 : 51-5.
7. Bedenne L, Faivere J, Boutron MC et al. Adenoma-Carcinoma Sequence or "De Novo" Carcinogenesis? Cancer 1992 ; 69 : 883-8.