-
1
-
-
38149091943
-
Adult nephrotic syndrome: non-specific strategies for treatment
-
Charlesworth J.A., Gracey D.M., and Pussell B.A. Adult nephrotic syndrome: non-specific strategies for treatment. Nephrology 3 (2008) 45-50
-
(2008)
Nephrology
, vol.3
, pp. 45-50
-
-
Charlesworth, J.A.1
Gracey, D.M.2
Pussell, B.A.3
-
3
-
-
0022724635
-
The hemodynamic basis of progressive renal disease
-
Dunn B.R., Anderson S., and Brenner B.M. The hemodynamic basis of progressive renal disease. Semin Nephrol 6 (1986) 122-138
-
(1986)
Semin Nephrol
, vol.6
, pp. 122-138
-
-
Dunn, B.R.1
Anderson, S.2
Brenner, B.M.3
-
4
-
-
0032512094
-
Pathophysiology of progressive nephropathies
-
Remuzzi G., and Bertani T. Pathophysiology of progressive nephropathies. N Engl J Med 339 (1998) 1448-1456
-
(1998)
N Engl J Med
, vol.339
, pp. 1448-1456
-
-
Remuzzi, G.1
Bertani, T.2
-
6
-
-
36148955109
-
Mechanisms of progression of chronic kidney disease
-
Fogo A.B. Mechanisms of progression of chronic kidney disease. Pediatr Nephrol 22 (2007) 2011-2022
-
(2007)
Pediatr Nephrol
, vol.22
, pp. 2011-2022
-
-
Fogo, A.B.1
-
7
-
-
54949158787
-
New insights into the mechanisms of fibrosis and sclerosis in diabetic nephropathy
-
Brosius III F.C. New insights into the mechanisms of fibrosis and sclerosis in diabetic nephropathy. Rev Endocr Metab Disord 9 (2008) 245-254
-
(2008)
Rev Endocr Metab Disord
, vol.9
, pp. 245-254
-
-
Brosius III, F.C.1
-
8
-
-
0024410794
-
Albuminuria reflects widespread vascular damage. The Steno hypothesis
-
Deckert T., Feldt-Rasmussen B., Borch-Johnsen K., Jensen T., and Kofoed-Enevoldsen A. Albuminuria reflects widespread vascular damage. The Steno hypothesis. Diabetologia 32 (1989) 219-226
-
(1989)
Diabetologia
, vol.32
, pp. 219-226
-
-
Deckert, T.1
Feldt-Rasmussen, B.2
Borch-Johnsen, K.3
Jensen, T.4
Kofoed-Enevoldsen, A.5
-
9
-
-
33746553765
-
Cross-talk between the kidney and the cardiovascular system
-
Amann K., Wanner C., and Ritz E. Cross-talk between the kidney and the cardiovascular system. J Am Soc Nephrol 17 (2006) 2112-2119
-
(2006)
J Am Soc Nephrol
, vol.17
, pp. 2112-2119
-
-
Amann, K.1
Wanner, C.2
Ritz, E.3
-
10
-
-
33746473003
-
Microalbuminuria and risk for cardiovascular disease: analysis of potential mechanisms
-
Stehouwer C.D., and Smulders Y.M. Microalbuminuria and risk for cardiovascular disease: analysis of potential mechanisms. J Am Soc Nephrol 17 (2006) 2106-2111
-
(2006)
J Am Soc Nephrol
, vol.17
, pp. 2106-2111
-
-
Stehouwer, C.D.1
Smulders, Y.M.2
-
11
-
-
33746523781
-
Microalbuminuria as an early marker for cardiovascular disease
-
de Zeeuw D., Parving H.H., and Henning R.H. Microalbuminuria as an early marker for cardiovascular disease. J Am Soc Nephrol 17 (2006) 2100-2105
-
(2006)
J Am Soc Nephrol
, vol.17
, pp. 2100-2105
-
-
de Zeeuw, D.1
Parving, H.H.2
Henning, R.H.3
-
12
-
-
0034044949
-
Glomerular hypertension, abnormal glomerular growth, and progression of renal diseases
-
Fogo A.B. Glomerular hypertension, abnormal glomerular growth, and progression of renal diseases. Kidney Int Suppl 75 (2000) S15-S21
-
(2000)
Kidney Int Suppl
, vol.75
-
-
Fogo, A.B.1
-
13
-
-
27844454420
-
Treatment of hypertension in chronic kidney disease
-
Toto R.D. Treatment of hypertension in chronic kidney disease. Semin Nephrol 25 (2005) 435-439
-
(2005)
Semin Nephrol
, vol.25
, pp. 435-439
-
-
Toto, R.D.1
-
14
-
-
26944503427
-
Renal protection in hypertensive patients: selection of antihypertensive therapy
-
Wenzel R.R. Renal protection in hypertensive patients: selection of antihypertensive therapy. Drugs 65 Suppl 2 (2005) 29-39
-
(2005)
Drugs
, vol.65
, Issue.SUPPL. 2
, pp. 29-39
-
-
Wenzel, R.R.1
-
15
-
-
34250812062
-
Canadian Hypertension Education Program. Treatment of hypertension in patients with nondiabetic chronic kidney disease
-
Ruzicka M., Burns K.D., Culleton B., and Tobe S.W. Canadian Hypertension Education Program. Treatment of hypertension in patients with nondiabetic chronic kidney disease. Can J Cardiol 23 (2007) 595-601
-
(2007)
Can J Cardiol
, vol.23
, pp. 595-601
-
-
Ruzicka, M.1
Burns, K.D.2
Culleton, B.3
Tobe, S.W.4
-
16
-
-
0028847815
-
Angiotensin II induces hypertrophy and hyperplasia in adult human mesangial cells
-
Orth S.R., Weinreich T., Bönisch S., Weih M., and Ritz E. Angiotensin II induces hypertrophy and hyperplasia in adult human mesangial cells. Exp Nephrol 3 (1995) 23-33
-
(1995)
Exp Nephrol
, vol.3
, pp. 23-33
-
-
Orth, S.R.1
Weinreich, T.2
Bönisch, S.3
Weih, M.4
Ritz, E.5
-
17
-
-
0027570187
-
Angiotensin II as a renal growth factor?
-
Wolf G., and Neilson E.G. Angiotensin II as a renal growth factor?. J Am Soc Nephrol 3 (1993) 1531-1540
-
(1993)
J Am Soc Nephrol
, vol.3
, pp. 1531-1540
-
-
Wolf, G.1
Neilson, E.G.2
-
18
-
-
0031049715
-
Modulation of plasminogen activator inhibitor-1 (PAI-1) in vivo: a new mechanism for the anti-fibrotic effect of renin-angiotensin inhibition
-
Oikawa T., Freeman M., Lo W., Vaughan D.E., and Fogo A. Modulation of plasminogen activator inhibitor-1 (PAI-1) in vivo: a new mechanism for the anti-fibrotic effect of renin-angiotensin inhibition. Kidney Int 51 (1997) 164-172
-
(1997)
Kidney Int
, vol.51
, pp. 164-172
-
-
Oikawa, T.1
Freeman, M.2
Lo, W.3
Vaughan, D.E.4
Fogo, A.5
-
19
-
-
0028987941
-
Transforming growth factor-b and angiotensin II: the missing link from glomerular hyperfiltration to glomerulosclerosis?
-
Ketteler M., Noble N.A., and Border W.A. Transforming growth factor-b and angiotensin II: the missing link from glomerular hyperfiltration to glomerulosclerosis?. Ann Rev Physiol 57 (1995) 279-295
-
(1995)
Ann Rev Physiol
, vol.57
, pp. 279-295
-
-
Ketteler, M.1
Noble, N.A.2
Border, W.A.3
-
20
-
-
58149247876
-
Mechanisms of kidney fibrosis and the role of anti-fibrotic therapy
-
Deelman L., and Sharma K. Mechanisms of kidney fibrosis and the role of anti-fibrotic therapy. Cur Op Nephrol Hypertens 18 (2009) 85-90
-
(2009)
Cur Op Nephrol Hypertens
, vol.18
, pp. 85-90
-
-
Deelman, L.1
Sharma, K.2
-
21
-
-
33644638349
-
Renal fibrosis: new insights into the pathogenesis and therapeutics
-
Mini review highlighting cellular and molecular pathways leading to renal fibrosis, and in particular detailing the fibrogenic action of TGF-β/Smad signalling as a novel target for therapeutic strategies in renal fibrosis.
-
Liu-Y. Renal fibrosis: new insights into the pathogenesis and therapeutics. Kidney Int 69 (2006) 213-217. Mini review highlighting cellular and molecular pathways leading to renal fibrosis, and in particular detailing the fibrogenic action of TGF-β/Smad signalling as a novel target for therapeutic strategies in renal fibrosis.
-
(2006)
Kidney Int
, vol.69
, pp. 213-217
-
-
Liu, Y.1
-
22
-
-
41749125671
-
Therapeutic strategies to slow chronic kidney disease progression
-
Wühl E., and Schaefer F. Therapeutic strategies to slow chronic kidney disease progression. Pediatr Nephrol 23 (2008) 705-716
-
(2008)
Pediatr Nephrol
, vol.23
, pp. 705-716
-
-
Wühl, E.1
Schaefer, F.2
-
23
-
-
34250754375
-
Novel therapeutic targets for diabetic nephropathy
-
Comprehensive review describing the interaction between metabolic and hemodynamic pathways in diabetic nephropathy, and providing an overview of interfering drugs currently used clinically as a first-line treatment or under current clinical development for the treatment of diabetic nephropathy.
-
Fukami K., Yamagishi S., Ueda S., and Okuda S. Novel therapeutic targets for diabetic nephropathy. Endocr Metab Immune Disord Drug Targets 7 (2007) 83-92. Comprehensive review describing the interaction between metabolic and hemodynamic pathways in diabetic nephropathy, and providing an overview of interfering drugs currently used clinically as a first-line treatment or under current clinical development for the treatment of diabetic nephropathy.
-
(2007)
Endocr Metab Immune Disord Drug Targets
, vol.7
, pp. 83-92
-
-
Fukami, K.1
Yamagishi, S.2
Ueda, S.3
Okuda, S.4
-
24
-
-
14644398528
-
Renal glomerular permselectivity and vascular endothelium
-
Camici M. Renal glomerular permselectivity and vascular endothelium. Biomed Pharmacother 59 (2005) 30-37
-
(2005)
Biomed Pharmacother
, vol.59
, pp. 30-37
-
-
Camici, M.1
-
25
-
-
85190694751
-
Endothelial dysfunction contributes to renal function-associated cardiovascular mortality in a population wit mild renal insufficiency: the Hoorn study
-
Stam F., van Guldener C., Becker A., Dekker J.M., Heine R.J., Bouter L.M., and Stehouwer C.D.A. Endothelial dysfunction contributes to renal function-associated cardiovascular mortality in a population wit mild renal insufficiency: the Hoorn study. J Am Soc Nephrol 61 (2006) 586-590
-
(2006)
J Am Soc Nephrol
, vol.61
, pp. 586-590
-
-
Stam, F.1
van Guldener, C.2
Becker, A.3
Dekker, J.M.4
Heine, R.J.5
Bouter, L.M.6
Stehouwer, C.D.A.7
-
26
-
-
39449084717
-
The endothelium as a target in renal diseases
-
Zoccali C. The endothelium as a target in renal diseases. J Nephrol 20 Suppl 12 (2007) S39-S44
-
(2007)
J Nephrol
, vol.20
, Issue.SUPPL. 12
-
-
Zoccali, C.1
-
27
-
-
34347350171
-
Chronic kidney disease. Effects of the cardiovascular system
-
Schiffrin E.L., Lipman M.L., and Mann J.F.E. Chronic kidney disease. Effects of the cardiovascular system. Circulation 116 (2007) 85-97
-
(2007)
Circulation
, vol.116
, pp. 85-97
-
-
Schiffrin, E.L.1
Lipman, M.L.2
Mann, J.F.E.3
-
28
-
-
50249110698
-
Endothelial dysfunction and chronic kidney disease: treatment options
-
Wu-Wong J.R. Endothelial dysfunction and chronic kidney disease: treatment options. Curr Opin Investig Drugs 9 (2008) 970-982
-
(2008)
Curr Opin Investig Drugs
, vol.9
, pp. 970-982
-
-
Wu-Wong, J.R.1
-
29
-
-
0035964954
-
Loss of caveolae, vascular dysfunction, and pulmonary defects in caveolin-1 gene-disrupted mice
-
Drab M., Verkade P., Elger M., Kasper M., Lohn M., Lauterbach B., Menne J., Lindschau C., Mende F., Luft F.C., et al. Loss of caveolae, vascular dysfunction, and pulmonary defects in caveolin-1 gene-disrupted mice. Science 293 (2001) 2449-2452
-
(2001)
Science
, vol.293
, pp. 2449-2452
-
-
Drab, M.1
Verkade, P.2
Elger, M.3
Kasper, M.4
Lohn, M.5
Lauterbach, B.6
Menne, J.7
Lindschau, C.8
Mende, F.9
Luft, F.C.10
-
30
-
-
33947323335
-
Disruption of endothelial caveolae is associated with impairment of both NO- as well as EDHF in acetylcholine-induced relaxation depending on their relative contribution in different vascular beds
-
Xu Y., Henning R.H., van der Want J.J.L., van Buiten A., van Gilst W.H., and Buikema H. Disruption of endothelial caveolae is associated with impairment of both NO- as well as EDHF in acetylcholine-induced relaxation depending on their relative contribution in different vascular beds. Life Sci 80 (2007) 1678-1685
-
(2007)
Life Sci
, vol.80
, pp. 1678-1685
-
-
Xu, Y.1
Henning, R.H.2
van der Want, J.J.L.3
van Buiten, A.4
van Gilst, W.H.5
Buikema, H.6
-
31
-
-
0038785720
-
Caveolin, caveolae, and endothelial cell function
-
Frank P.G., Woodman S.E., Park D.S., and Lisanti M.P. Caveolin, caveolae, and endothelial cell function. Arterioscler Thromb Vasc Biol 23 (2003) 1161-1168
-
(2003)
Arterioscler Thromb Vasc Biol
, vol.23
, pp. 1161-1168
-
-
Frank, P.G.1
Woodman, S.E.2
Park, D.S.3
Lisanti, M.P.4
-
32
-
-
0032500605
-
Rapid mechanotransduction in situ at the luminal cell surface of the microvascular endothelium and its caveolae
-
Rizzo V., Sung A., and Schnitzer J.E. Rapid mechanotransduction in situ at the luminal cell surface of the microvascular endothelium and its caveolae. J Biol Chem 273 (1998) 26323-26329
-
(1998)
J Biol Chem
, vol.273
, pp. 26323-26329
-
-
Rizzo, V.1
Sung, A.2
Schnitzer, J.E.3
-
33
-
-
33947261951
-
Mechanical stress-initiated signal transduction in vascular smooth muscle cells in vitro and in vivo
-
Li C., and Xu Q. Mechanical stress-initiated signal transduction in vascular smooth muscle cells in vitro and in vivo. Cell Signal 19 (2007) 881-891
-
(2007)
Cell Signal
, vol.19
, pp. 881-891
-
-
Li, C.1
Xu, Q.2
-
34
-
-
62349131464
-
-
Xu Y, Henning RH, Sandovici M, van der Want JJL, van Gilst WH, Buikema H: Enhanced myogenic constriction of mesenteric artery in heart failure relates to decreased smooth muscle cell caveolae number and altered AT1-receptor and EGF-receptor function. Eur J Heart Fail 2009, in press. Animal study providing evidence that decreased vascular smooth muscle cell caveolae number may be implicated in enhanced myogenic response in small mesenteric arteries of rats with experimentally induced heart failure by affecting constitutive EGF-receptor activity. The results of this study suggest a mechanism of derailments in vasomotor autoregulation as a consequence of caveolar changes in pathophysiological conditions via basic alterations in receptor function. It may challenge future studies for investigation of alterations in caveolar changes underlying impaired autoregulation in renal vasculature in pathophysiological conditions of kidney disease
-
1-receptor and EGF-receptor function. Eur J Heart Fail 2009, in press. Animal study providing evidence that decreased vascular smooth muscle cell caveolae number may be implicated in enhanced myogenic response in small mesenteric arteries of rats with experimentally induced heart failure by affecting constitutive EGF-receptor activity. The results of this study suggest a mechanism of derailments in vasomotor autoregulation as a consequence of caveolar changes in pathophysiological conditions via basic alterations in receptor function. It may challenge future studies for investigation of alterations in caveolar changes underlying impaired autoregulation in renal vasculature in pathophysiological conditions of kidney disease.
-
-
-
-
35
-
-
34548457220
-
Arterial remodeling and plasma volume expansion in caveolin-1-deficient mice
-
Albinsson S., Shakirova Y., Rippe A., Baumgarten M., Rosengren B.I., Rippe C., Hallmann R., Hellstrand P., Rippe B., and Swärd K. Arterial remodeling and plasma volume expansion in caveolin-1-deficient mice. Am J Physiol Regul Integr Comp Physiol 293 (2007) R1222-R1231
-
(2007)
Am J Physiol Regul Integr Comp Physiol
, vol.293
-
-
Albinsson, S.1
Shakirova, Y.2
Rippe, A.3
Baumgarten, M.4
Rosengren, B.I.5
Rippe, C.6
Hallmann, R.7
Hellstrand, P.8
Rippe, B.9
Swärd, K.10
-
36
-
-
33847729462
-
Caveolin-1 abolishment attenuates the myogenic response in murine cerebral arteries
-
••] in that both studies underline the importance of caveolae involved in normal autoregulation of vascular tone on the one hand. While on the other hand, the discrepancy between both studies at the same time underlines the importance of caveolae as a structural entity as well as presence of cav-1 as a direct regulatory protein. Above all, however, such studies should challenge future studies for investigation of alterations in caveolar changes underlying impaired autoregulation in renal vasculature in pathophysiological conditions of kidney disease.
-
••] in that both studies underline the importance of caveolae involved in normal autoregulation of vascular tone on the one hand. While on the other hand, the discrepancy between both studies at the same time underlines the importance of caveolae as a structural entity as well as presence of cav-1 as a direct regulatory protein. Above all, however, such studies should challenge future studies for investigation of alterations in caveolar changes underlying impaired autoregulation in renal vasculature in pathophysiological conditions of kidney disease.
-
(2007)
Am J Physiol Heart Circ Physiol
, vol.292
-
-
Adebiyi, A.1
Zhao, G.2
Cheranov, S.Y.3
Ahmed, A.4
Jaggar, J.H.5
-
37
-
-
0032898637
-
Altered renal hemodynamics and impaired myogenic responses in the fawn-hooded rat
-
Van Dokkum R.P., Sun C.W., Provoost A.P., Jacob H.J., and Roman R.J. Altered renal hemodynamics and impaired myogenic responses in the fawn-hooded rat. Am J Physiol Regul Integr Comp Physiol 276 (1999) R855-R863
-
(1999)
Am J Physiol Regul Integr Comp Physiol
, vol.276
-
-
Van Dokkum, R.P.1
Sun, C.W.2
Provoost, A.P.3
Jacob, H.J.4
Roman, R.J.5
-
38
-
-
43049124748
-
Caveolae and endothelial dysfunction: filling the caves in cardiovascular disease
-
Xu Y., Buikema H., van Gilst W.H., and Henning R.H. Caveolae and endothelial dysfunction: filling the caves in cardiovascular disease. Eur J Pharmacol 585 (2008) 256-260
-
(2008)
Eur J Pharmacol
, vol.585
, pp. 256-260
-
-
Xu, Y.1
Buikema, H.2
van Gilst, W.H.3
Henning, R.H.4
-
39
-
-
0035096636
-
Control of glomerular capillary development by growth factors/receptor kinases
-
Robert B., and Abrahamson D.R. Control of glomerular capillary development by growth factors/receptor kinases. Pediatr Nephrol 16 (2001) 294-301
-
(2001)
Pediatr Nephrol
, vol.16
, pp. 294-301
-
-
Robert, B.1
Abrahamson, D.R.2
-
40
-
-
0037631361
-
Neutralization of circulating vascular endothelial growth factor (VEGF) by anti-VEGF antibodies and soluble VEGF receptor 1 (sFit-1) induces proteinuria
-
Sugimoto H., Hamano Y., Charytan D., Cosgrove D., Kieran M., Sudhakor A., and Kalluri R.I. Neutralization of circulating vascular endothelial growth factor (VEGF) by anti-VEGF antibodies and soluble VEGF receptor 1 (sFit-1) induces proteinuria. J Biochem 278 (2003) 12605-12608
-
(2003)
J Biochem
, vol.278
, pp. 12605-12608
-
-
Sugimoto, H.1
Hamano, Y.2
Charytan, D.3
Cosgrove, D.4
Kieran, M.5
Sudhakor, A.6
Kalluri, R.I.7
-
41
-
-
1542349097
-
Angiopoietin 1 and vascular endothelial growth factor modulate human glomerular endothelial cell barrier properties
-
Satchell S.C., Anderson K.L., and Mathieson P.W. Angiopoietin 1 and vascular endothelial growth factor modulate human glomerular endothelial cell barrier properties. J Am Soc Nephrol 15 (2004) 566-574
-
(2004)
J Am Soc Nephrol
, vol.15
, pp. 566-574
-
-
Satchell, S.C.1
Anderson, K.L.2
Mathieson, P.W.3
-
42
-
-
60549110938
-
Lipid rafts and caveolae in signaling by growth factor receptors
-
De Laurentiis A., Donovan L., and Arcaro A. Lipid rafts and caveolae in signaling by growth factor receptors. Open Biochem J 1 (2007) 12-32
-
(2007)
Open Biochem J
, vol.1
, pp. 12-32
-
-
De Laurentiis, A.1
Donovan, L.2
Arcaro, A.3
-
44
-
-
34047251876
-
Caveolin-1 deficient mice have increased tumor microvascular permeability, angiogenesis, and growth
-
Lin M.I., Yu J., Murata T., and Sessa W.C. Caveolin-1 deficient mice have increased tumor microvascular permeability, angiogenesis, and growth. Cancer Res 67 (2007) 2849-2856
-
(2007)
Cancer Res
, vol.67
, pp. 2849-2856
-
-
Lin, M.I.1
Yu, J.2
Murata, T.3
Sessa, W.C.4
-
45
-
-
0036087482
-
VEGF-induced mobilization of caveolae and increase in permeability of endothelial cells
-
Chen J., Braet, Brodsky, Weinstein T., Romanov V., Noiri E., and Goligorsky M.S. VEGF-induced mobilization of caveolae and increase in permeability of endothelial cells. Am J Physiol Cell Physiol 282 (2002) C1053-C1063
-
(2002)
Am J Physiol Cell Physiol
, vol.282
-
-
Chen, J.1
Braet2
Brodsky3
Weinstein, T.4
Romanov, V.5
Noiri, E.6
Goligorsky, M.S.7
-
46
-
-
70349822208
-
Compartmentalization of Redox Signalling through NADPH Oxidase-derived ROS
-
Epub ahead of print
-
Ushio-Fukai M. Compartmentalization of Redox Signalling through NADPH Oxidase-derived ROS. Antioxid Redox Signal (2008) Epub ahead of print
-
(2008)
Antioxid Redox Signal
-
-
Ushio-Fukai, M.1
-
47
-
-
49949102465
-
TGFβ-induced RhoA activation and fibronectin production in mesangial cells require caveolae
-
Using primary rat mesangial cells, the authors show the requirement of caveolae and caveolin-1 interaction with RhoA in TGF-β-induced fibronectin upregulation. Such results may highlight caveolae as a potential interventional target for the treatment of fibrotic renal disease.
-
Peng F., Zhang B., Wu D., Ingram A.J., Gao B., and Krepinsky J.C. TGFβ-induced RhoA activation and fibronectin production in mesangial cells require caveolae. Am J Physiol Renal Physiol 295 (2008) F153-F164. Using primary rat mesangial cells, the authors show the requirement of caveolae and caveolin-1 interaction with RhoA in TGF-β-induced fibronectin upregulation. Such results may highlight caveolae as a potential interventional target for the treatment of fibrotic renal disease.
-
(2008)
Am J Physiol Renal Physiol
, vol.295
-
-
Peng, F.1
Zhang, B.2
Wu, D.3
Ingram, A.J.4
Gao, B.5
Krepinsky, J.C.6
-
48
-
-
57749189130
-
Caveolae and transcytosis in endothelial cells: role in atherosclerosis
-
Frank P.G., Pavlides S., and Lisanti M.P. Caveolae and transcytosis in endothelial cells: role in atherosclerosis. Cell Tissue Res 335 (2009) 41-47
-
(2009)
Cell Tissue Res
, vol.335
, pp. 41-47
-
-
Frank, P.G.1
Pavlides, S.2
Lisanti, M.P.3
-
49
-
-
0037131313
-
Microvascular hyperpermeability in caveolin-1 (-/-) knockout mice: treatment with a specific nitric-oxide synthase inhibitor, L-NAME, restores normal microvascular permeability in Cav-1 null mice
-
Schubert W., Frank P.G., Woodman S.E., Hoyogo H., Cohen D.E., Chow C.W., and Lisanti M.P. Microvascular hyperpermeability in caveolin-1 (-/-) knockout mice: treatment with a specific nitric-oxide synthase inhibitor, L-NAME, restores normal microvascular permeability in Cav-1 null mice. J Biol Chem 277 (2002) 40091-40098
-
(2002)
J Biol Chem
, vol.277
, pp. 40091-40098
-
-
Schubert, W.1
Frank, P.G.2
Woodman, S.E.3
Hoyogo, H.4
Cohen, D.E.5
Chow, C.W.6
Lisanti, M.P.7
-
50
-
-
0026769237
-
Endothelial dysfunction in chronic congestive heart failure
-
Drexler H., Hayoz D., Munzel T., Hornig B., Just H., Brunner H.R., and Zelis R. Endothelial dysfunction in chronic congestive heart failure. Am J Cardiol 69 (1992) 1596-1601
-
(1992)
Am J Cardiol
, vol.69
, pp. 1596-1601
-
-
Drexler, H.1
Hayoz, D.2
Munzel, T.3
Hornig, B.4
Just, H.5
Brunner, H.R.6
Zelis, R.7
-
51
-
-
0036082492
-
Relationships between caveolae and eNOS: everything in proximity and the proximity of everything
-
Goligorsky M.S., Hong L., Brodsky S., and Chen J. Relationships between caveolae and eNOS: everything in proximity and the proximity of everything. Am J Physiol Renal Physiol 52 (2002) F1-F10
-
(2002)
Am J Physiol Renal Physiol
, vol.52
-
-
Goligorsky, M.S.1
Hong, L.2
Brodsky, S.3
Chen, J.4
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