-
1
-
-
50149086108
-
Diversity of degradation signals in the ubiquitin-proteasome system
-
Ravid T., and Hochstrasser M. Diversity of degradation signals in the ubiquitin-proteasome system. Nat Rev Mol Cell Biol 9 (2008) 679-690
-
(2008)
Nat Rev Mol Cell Biol
, vol.9
, pp. 679-690
-
-
Ravid, T.1
Hochstrasser, M.2
-
2
-
-
56949095293
-
The ubiquitin-proteasome system in cardiac dysfunction
-
An extensive updated review on cardiac ubiquitin-proteasome system and its role in cardiac disease.
-
Mearini G., Schlossarek S., Willis M.S., and Carrier L. The ubiquitin-proteasome system in cardiac dysfunction. Biochim Biophys Acta 1782 (2008) 749-763. An extensive updated review on cardiac ubiquitin-proteasome system and its role in cardiac disease.
-
(2008)
Biochim Biophys Acta
, vol.1782
, pp. 749-763
-
-
Mearini, G.1
Schlossarek, S.2
Willis, M.S.3
Carrier, L.4
-
3
-
-
33745726659
-
The ubiquitin-proteasome system in cardiac physiology and pathology
-
Powell S.R. The ubiquitin-proteasome system in cardiac physiology and pathology. Am J Physiol Heart Circ Physiol 291 (2006) H1-H19
-
(2006)
Am J Physiol Heart Circ Physiol
, vol.291
-
-
Powell, S.R.1
-
4
-
-
43049092644
-
Understanding proteasome assembly and regulation: importance to cardiovascular medicine
-
Young G.W., Wang Y., and Ping P. Understanding proteasome assembly and regulation: importance to cardiovascular medicine. Trends Cardiovasc Med 18 (2008) 93-98
-
(2008)
Trends Cardiovasc Med
, vol.18
, pp. 93-98
-
-
Young, G.W.1
Wang, Y.2
Ping, P.3
-
5
-
-
61649083080
-
Contrasting proteome biology and functional heterogeneity of the 20S proteasome complexes in mammalian tissues
-
First proteomic report on organ-specific responses to proteasome inhibition. Proteomic studies reveal significant heterogeneity in the structure and function of the proteasomes in the heart and liver.
-
Gomes A.V., Young G.W., Wang Y., Zong C., Eghbali M., Drews O., Lu H., Stefani E., and Ping P. Contrasting proteome biology and functional heterogeneity of the 20S proteasome complexes in mammalian tissues. Mol Cell Proteomics 8 (2009) 302-315. First proteomic report on organ-specific responses to proteasome inhibition. Proteomic studies reveal significant heterogeneity in the structure and function of the proteasomes in the heart and liver.
-
(2009)
Mol Cell Proteomics
, vol.8
, pp. 302-315
-
-
Gomes, A.V.1
Young, G.W.2
Wang, Y.3
Zong, C.4
Eghbali, M.5
Drews, O.6
Lu, H.7
Stefani, E.8
Ping, P.9
-
6
-
-
0035839573
-
Oxidative modification and inactivation of the proteasome during coronary occlusion/reperfusion
-
Bulteau A.L., Lundberg K.C., Humphries K.M., Sadek H.A., Szweda P.A., Friguet B., and Szweda L.I. Oxidative modification and inactivation of the proteasome during coronary occlusion/reperfusion. J Biol Chem 276 (2001) 30057-30063
-
(2001)
J Biol Chem
, vol.276
, pp. 30057-30063
-
-
Bulteau, A.L.1
Lundberg, K.C.2
Humphries, K.M.3
Sadek, H.A.4
Szweda, P.A.5
Friguet, B.6
Szweda, L.I.7
-
7
-
-
17644375852
-
Oxidized and ubiquitinated proteins may predict recovery of postischemic cardiac function: essential role of the proteasome
-
Powell S.R., Wang P., Katzeff H., Shringarpure R., Teoh C., Khaliulin I., Das D.K., Davies K.J., and Schwalb H. Oxidized and ubiquitinated proteins may predict recovery of postischemic cardiac function: essential role of the proteasome. Antioxid Redox Signal 7 (2005) 538-546
-
(2005)
Antioxid Redox Signal
, vol.7
, pp. 538-546
-
-
Powell, S.R.1
Wang, P.2
Katzeff, H.3
Shringarpure, R.4
Teoh, C.5
Khaliulin, I.6
Das, D.K.7
Davies, K.J.8
Schwalb, H.9
-
8
-
-
27144441722
-
Oxidative modification of proteasome: identification of an oxidation-sensitive subunit in 26 S proteasome
-
Ishii T., Sakurai T., Usami H., and Uchida K. Oxidative modification of proteasome: identification of an oxidation-sensitive subunit in 26 S proteasome. Biochemistry 44 (2005) 13893-13901
-
(2005)
Biochemistry
, vol.44
, pp. 13893-13901
-
-
Ishii, T.1
Sakurai, T.2
Usami, H.3
Uchida, K.4
-
9
-
-
21644442676
-
Cardioprotection with palm tocotrienol: antioxidant activity of tocotrienol is linked with its ability to stabilize proteasomes
-
Das S., Powell S.R., Wang P., Divald A., Nesaretnam K., Tosaki A., Cordis G.A., Maulik N., and Das D.K. Cardioprotection with palm tocotrienol: antioxidant activity of tocotrienol is linked with its ability to stabilize proteasomes. Am J Physiol Heart Circ Physiol 289 (2005) H361-367
-
(2005)
Am J Physiol Heart Circ Physiol
, vol.289
-
-
Das, S.1
Powell, S.R.2
Wang, P.3
Divald, A.4
Nesaretnam, K.5
Tosaki, A.6
Cordis, G.A.7
Maulik, N.8
Das, D.K.9
-
10
-
-
28744453336
-
Proteasome mediates removal of proteins oxidized during myocardial ischemia
-
Divald A., and Powell S.R. Proteasome mediates removal of proteins oxidized during myocardial ischemia. Free Radic Biol Med 40 (2006) 156-164
-
(2006)
Free Radic Biol Med
, vol.40
, pp. 156-164
-
-
Divald, A.1
Powell, S.R.2
-
11
-
-
33747770049
-
Inactivation of the proteasome by 4-hydroxy-2-nonenal is site specific and dependent on 20S proteasome subtypes
-
Farout L., Mary J., Vinh J., Szweda L.I., and Friguet B. Inactivation of the proteasome by 4-hydroxy-2-nonenal is site specific and dependent on 20S proteasome subtypes. Arch Biochem Biophys 453 (2006) 135-142
-
(2006)
Arch Biochem Biophys
, vol.453
, pp. 135-142
-
-
Farout, L.1
Mary, J.2
Vinh, J.3
Szweda, L.I.4
Friguet, B.5
-
12
-
-
39149124870
-
Oxidative injury induces selective rather than global inhibition of proteasomal activity
-
This study demonstrates that a specific rather than global proteasome inhibition occurs in the heart during ischemia/reperfusion injury.
-
Gurusamy N., Goswami S., Malik G., and Das D.K. Oxidative injury induces selective rather than global inhibition of proteasomal activity. J Mol Cell Cardiol 44 (2008) 419-428. This study demonstrates that a specific rather than global proteasome inhibition occurs in the heart during ischemia/reperfusion injury.
-
(2008)
J Mol Cell Cardiol
, vol.44
, pp. 419-428
-
-
Gurusamy, N.1
Goswami, S.2
Malik, G.3
Das, D.K.4
-
13
-
-
30544446127
-
Depression of proteasome activities during the progression of cardiac dysfunction in pressure-overloaded heart of mice
-
Tsukamoto O., Minamino T., Okada K., Shintani Y., Takashima S., Kato H., Liao Y., Okazaki H., Asai M., Hirata A., et al. Depression of proteasome activities during the progression of cardiac dysfunction in pressure-overloaded heart of mice. Biochem Biophys Res Commun 340 (2006) 1125-1133
-
(2006)
Biochem Biophys Res Commun
, vol.340
, pp. 1125-1133
-
-
Tsukamoto, O.1
Minamino, T.2
Okada, K.3
Shintani, Y.4
Takashima, S.5
Kato, H.6
Liao, Y.7
Okazaki, H.8
Asai, M.9
Hirata, A.10
-
14
-
-
0037328263
-
Hyperubiquitination of proteins in dilated cardiomyopathy
-
Weekes J., Morrison K., Mullen A., Wait R., Barton P., and Dunn M.J. Hyperubiquitination of proteins in dilated cardiomyopathy. Proteomics 3 (2003) 208-216
-
(2003)
Proteomics
, vol.3
, pp. 208-216
-
-
Weekes, J.1
Morrison, K.2
Mullen, A.3
Wait, R.4
Barton, P.5
Dunn, M.J.6
-
15
-
-
48749126678
-
Elevated p53 expression is associated with dysregulation of the ubiquitin-proteasome system in dilated cardiomyopathy
-
Birks E.J., Latif N., Enesa K., Folkvang T., Luong le A., Sarathchandra P., Khan M., Ovaa H., Terracciano C.M., Barton P.J., et al. Elevated p53 expression is associated with dysregulation of the ubiquitin-proteasome system in dilated cardiomyopathy. Cardiovasc Res 79 (2008) 472-480
-
(2008)
Cardiovasc Res
, vol.79
, pp. 472-480
-
-
Birks, E.J.1
Latif, N.2
Enesa, K.3
Folkvang, T.4
Luong le, A.5
Sarathchandra, P.6
Khan, M.7
Ovaa, H.8
Terracciano, C.M.9
Barton, P.J.10
-
16
-
-
33750543772
-
Activation of the cardiac proteasome during pressure overload promotes ventricular hypertrophy
-
Depre C., Wang Q., Yan L., Hedhli N., Peter P., Chen L., Hong C., Hittinger L., Ghaleh B., Sadoshima J., et al. Activation of the cardiac proteasome during pressure overload promotes ventricular hypertrophy. Circulation 114 (2006) 1821-1828
-
(2006)
Circulation
, vol.114
, pp. 1821-1828
-
-
Depre, C.1
Wang, Q.2
Yan, L.3
Hedhli, N.4
Peter, P.5
Chen, L.6
Hong, C.7
Hittinger, L.8
Ghaleh, B.9
Sadoshima, J.10
-
17
-
-
33748581280
-
Enhanced ubiquitination of cytoskeletal proteins in pressure overloaded myocardium is accompanied by changes in specific E3 ligases
-
Balasubramanian S., Mani S., Shiraishi H., Johnston R.K., Yamane K., Willey C.D., Cooper G.T., Tuxworth W.J., and Kuppuswamy D. Enhanced ubiquitination of cytoskeletal proteins in pressure overloaded myocardium is accompanied by changes in specific E3 ligases. J Mol Cell Cardiol 41 (2006) 669-679
-
(2006)
J Mol Cell Cardiol
, vol.41
, pp. 669-679
-
-
Balasubramanian, S.1
Mani, S.2
Shiraishi, H.3
Johnston, R.K.4
Yamane, K.5
Willey, C.D.6
Cooper, G.T.7
Tuxworth, W.J.8
Kuppuswamy, D.9
-
18
-
-
43549098451
-
Potential role of the ubiquitin-proteasome system in atherosclerosis: aspects of a protein quality disease
-
Herrmann J., Soares S.M., Lerman L.O., and Lerman A. Potential role of the ubiquitin-proteasome system in atherosclerosis: aspects of a protein quality disease. J Am Coll Cardiol 51 (2008) 2003-2010
-
(2008)
J Am Coll Cardiol
, vol.51
, pp. 2003-2010
-
-
Herrmann, J.1
Soares, S.M.2
Lerman, L.O.3
Lerman, A.4
-
19
-
-
37349073383
-
Chronic proteasome inhibition contributes to coronary atherosclerosis
-
Herrmann J., Saguner A.M., Versari D., Peterson T.E., Chade A., Olson M., Lerman L.O., and Lerman A. Chronic proteasome inhibition contributes to coronary atherosclerosis. Circ Res 101 (2007) 865-874
-
(2007)
Circ Res
, vol.101
, pp. 865-874
-
-
Herrmann, J.1
Saguner, A.M.2
Versari, D.3
Peterson, T.E.4
Chade, A.5
Olson, M.6
Lerman, L.O.7
Lerman, A.8
-
20
-
-
3442898375
-
The ubiquitin-proteasome pathway is a new partner for the control of insulin signaling
-
Rome S., Meugnier E., and Vidal H. The ubiquitin-proteasome pathway is a new partner for the control of insulin signaling. Curr Opin Clin Nutr Metab Care 7 (2004) 249-254
-
(2004)
Curr Opin Clin Nutr Metab Care
, vol.7
, pp. 249-254
-
-
Rome, S.1
Meugnier, E.2
Vidal, H.3
-
21
-
-
22744459667
-
Is insulin signaling molecules misguided in diabetes for ubiquitin-proteasome mediated degradation?
-
Balasubramanyam M., Sampathkumar R., and Mohan V. Is insulin signaling molecules misguided in diabetes for ubiquitin-proteasome mediated degradation?. Mol Cell Biochem 275 (2005) 117-125
-
(2005)
Mol Cell Biochem
, vol.275
, pp. 117-125
-
-
Balasubramanyam, M.1
Sampathkumar, R.2
Mohan, V.3
-
22
-
-
54349086813
-
Cardiac muscle protein catabolism in diabetes mellitus: activation of the ubiquitin-proteasome system by insulin deficiency
-
This paper describes diabetes-induced proteasomal degradation of cardiac proteins in streptozotocin-induced diabetic mice.
-
Hu J., Klein J.D., Du J., and Wang X.H. Cardiac muscle protein catabolism in diabetes mellitus: activation of the ubiquitin-proteasome system by insulin deficiency. Endocrinology 149 (2008) 5384-5390. This paper describes diabetes-induced proteasomal degradation of cardiac proteins in streptozotocin-induced diabetic mice.
-
(2008)
Endocrinology
, vol.149
, pp. 5384-5390
-
-
Hu, J.1
Klein, J.D.2
Du, J.3
Wang, X.H.4
-
23
-
-
40649101141
-
Upregulation of myocardial 11S-activated proteasome in experimental hyperglycemia
-
Powell S.R., Samuel S.M., Wang P., Divald A., Thirunavukkarasu M., Koneru S., Wang X., and Maulik N. Upregulation of myocardial 11S-activated proteasome in experimental hyperglycemia. J Mol Cell Cardiol 44 (2008) 618-621
-
(2008)
J Mol Cell Cardiol
, vol.44
, pp. 618-621
-
-
Powell, S.R.1
Samuel, S.M.2
Wang, P.3
Divald, A.4
Thirunavukkarasu, M.5
Koneru, S.6
Wang, X.7
Maulik, N.8
-
24
-
-
58149115206
-
REGgamma, a proteasome activator and beyond?
-
Mao I., Liu J., Li X., and Luo H. REGgamma, a proteasome activator and beyond?. Cell Mol Life Sci 65 (2008) 3971-3980
-
(2008)
Cell Mol Life Sci
, vol.65
, pp. 3971-3980
-
-
Mao, I.1
Liu, J.2
Li, X.3
Luo, H.4
-
25
-
-
50249092779
-
Development of proteasome inhibitors in oncology and autoimmune diseases
-
Bennett M.K., and Kirk C.J. Development of proteasome inhibitors in oncology and autoimmune diseases. Curr Opin Drug Discov Dev 11 (2008) 616-625
-
(2008)
Curr Opin Drug Discov Dev
, vol.11
, pp. 616-625
-
-
Bennett, M.K.1
Kirk, C.J.2
-
26
-
-
45749118333
-
The potential of proteasome inhibitors in cancer therapy
-
Sterz J., von Metzler I., Hahne J.C., Lamottke B., Rademacher J., Heider U., Terpos E., and Sezer O. The potential of proteasome inhibitors in cancer therapy. Expert Opin Investig Drugs 17 (2008) 879-895
-
(2008)
Expert Opin Investig Drugs
, vol.17
, pp. 879-895
-
-
Sterz, J.1
von Metzler, I.2
Hahne, J.C.3
Lamottke, B.4
Rademacher, J.5
Heider, U.6
Terpos, E.7
Sezer, O.8
-
27
-
-
34447123834
-
Unexpected cardiotoxicity in haematological bortezomib treated patients
-
Enrico O., Gabriele B., Nadia C., Sara G., Daniele V., Giulia C., Antonio S., and Mario P. Unexpected cardiotoxicity in haematological bortezomib treated patients. Br J Haematol 138 (2007) 396-397
-
(2007)
Br J Haematol
, vol.138
, pp. 396-397
-
-
Enrico, O.1
Gabriele, B.2
Nadia, C.3
Sara, G.4
Daniele, V.5
Giulia, C.6
Antonio, S.7
Mario, P.8
-
28
-
-
56649108096
-
Acute severe cardiac failure in a myeloma patient due to proteasome inhibitor bortezomib
-
Hacihanefioglu A., Tarkun P., and Gonullu E. Acute severe cardiac failure in a myeloma patient due to proteasome inhibitor bortezomib. Int J Hematol 88 (2008) 219-222
-
(2008)
Int J Hematol
, vol.88
, pp. 219-222
-
-
Hacihanefioglu, A.1
Tarkun, P.2
Gonullu, E.3
-
29
-
-
0037370653
-
Proteasome inhibition ablates activation of NF-kappa B in myocardial reperfusion and reduces reperfusion injury
-
Pye J., Ardeshirpour F., McCain A., Bellinger D.A., Merricks E., Adams J., Elliott P.J., Pien C., Fischer T.H., Baldwin Jr. A.S., et al. Proteasome inhibition ablates activation of NF-kappa B in myocardial reperfusion and reduces reperfusion injury. Am J Physiol Heart Circ Physiol 284 (2003) H919-H926
-
(2003)
Am J Physiol Heart Circ Physiol
, vol.284
-
-
Pye, J.1
Ardeshirpour, F.2
McCain, A.3
Bellinger, D.A.4
Merricks, E.5
Adams, J.6
Elliott, P.J.7
Pien, C.8
Fischer, T.H.9
Baldwin Jr., A.S.10
-
30
-
-
34250368734
-
Proteasome inhibition attenuates infarct size and preserves cardiac function in a murine model of myocardial ischemia-reperfusion injury
-
Shows proteasome inhibition reduces infarct size, attenuates myocardial inflammatory response, and preserves regional myocardial function in a murine infarct model.
-
Stansfield W.E., Moss N.C., Willis M.S., Tang R., and Selzman C.H. Proteasome inhibition attenuates infarct size and preserves cardiac function in a murine model of myocardial ischemia-reperfusion injury. Ann Thorac Surg 84 (2007) 120-125. Shows proteasome inhibition reduces infarct size, attenuates myocardial inflammatory response, and preserves regional myocardial function in a murine infarct model.
-
(2007)
Ann Thorac Surg
, vol.84
, pp. 120-125
-
-
Stansfield, W.E.1
Moss, N.C.2
Willis, M.S.3
Tang, R.4
Selzman, C.H.5
-
31
-
-
27144469875
-
Proteasome degradation of GRK2 during ischemia and ventricular tachyarrhythmias in a canine model of myocardial infarction
-
Yu X., Huang S., Patterson E., Garrett M.W., Kaufman K.M., Metcalf J.P., Zhu M., Dunn S.T., and Kem D.C. Proteasome degradation of GRK2 during ischemia and ventricular tachyarrhythmias in a canine model of myocardial infarction. Am J Physiol Heart Circ Physiol 289 (2005) H1960-H1967
-
(2005)
Am J Physiol Heart Circ Physiol
, vol.289
-
-
Yu, X.1
Huang, S.2
Patterson, E.3
Garrett, M.W.4
Kaufman, K.M.5
Metcalf, J.P.6
Zhu, M.7
Dunn, S.T.8
Kem, D.C.9
-
32
-
-
41549114524
-
Proteasome inhibition 1 h following ischemia protects GRK2 and prevents malignant ventricular tachyarrhythmias and SCD in a model of myocardial infarction
-
The authors show proteasome inhibition following myocardial ischemia suppresses fatal ventricular tachyarrhythmias, possibly by protecting against the loss of GRK2, in a canine model of myocardial infarction.
-
Huang S., Patterson E., Yu X., Garrett M.W., De Aos I., and Kem D.C. Proteasome inhibition 1 h following ischemia protects GRK2 and prevents malignant ventricular tachyarrhythmias and SCD in a model of myocardial infarction. Am J Physiol Heart Circ Physiol 294 (2008) H1298-H1303. The authors show proteasome inhibition following myocardial ischemia suppresses fatal ventricular tachyarrhythmias, possibly by protecting against the loss of GRK2, in a canine model of myocardial infarction.
-
(2008)
Am J Physiol Heart Circ Physiol
, vol.294
-
-
Huang, S.1
Patterson, E.2
Yu, X.3
Garrett, M.W.4
De Aos, I.5
Kem, D.C.6
-
34
-
-
0034649303
-
Ischemic inactivation of G protein-coupled receptor kinase and altered desensitization of canine cardiac beta-adrenergic receptors
-
Yu X., Zhang M., Kyker K., Patterson E., Benovic J.L., and Kem D.C. Ischemic inactivation of G protein-coupled receptor kinase and altered desensitization of canine cardiac beta-adrenergic receptors. Circulation 102 (2000) 2535-2540
-
(2000)
Circulation
, vol.102
, pp. 2535-2540
-
-
Yu, X.1
Zhang, M.2
Kyker, K.3
Patterson, E.4
Benovic, J.L.5
Kem, D.C.6
-
35
-
-
0032567351
-
Degradation of the G protein-coupled receptor kinase 2 by the proteasome pathway
-
Penela P., Ruiz-Gomez A., Castano J.G., and Mayor Jr. F. Degradation of the G protein-coupled receptor kinase 2 by the proteasome pathway. J Biol Chem 273 (1998) 35238-35244
-
(1998)
J Biol Chem
, vol.273
, pp. 35238-35244
-
-
Penela, P.1
Ruiz-Gomez, A.2
Castano, J.G.3
Mayor Jr., F.4
-
36
-
-
0035811062
-
Activation of NF-kappa B is required for hypertrophic growth of primary rat neonatal ventricular cardiomyocytes
-
Purcell N.H., Tang G., Yu C., Mercurio F., DiDonato J.A., and Lin A. Activation of NF-kappa B is required for hypertrophic growth of primary rat neonatal ventricular cardiomyocytes. Proc Natl Acad Sci U S A 98 (2001) 6668-6673
-
(2001)
Proc Natl Acad Sci U S A
, vol.98
, pp. 6668-6673
-
-
Purcell, N.H.1
Tang, G.2
Yu, C.3
Mercurio, F.4
DiDonato, J.A.5
Lin, A.6
-
37
-
-
48049083532
-
Blockade of NF-kappaB using IkappaB alpha dominant-negative mice ameliorates cardiac hypertrophy in myotrophin-overexpressed transgenic mice
-
Young D., Popovic Z.B., Jones W.K., and Gupta S. Blockade of NF-kappaB using IkappaB alpha dominant-negative mice ameliorates cardiac hypertrophy in myotrophin-overexpressed transgenic mice. J Mol Biol 381 (2008) 559-568
-
(2008)
J Mol Biol
, vol.381
, pp. 559-568
-
-
Young, D.1
Popovic, Z.B.2
Jones, W.K.3
Gupta, S.4
-
38
-
-
38549105011
-
Suppression of cardiomyocyte hypertrophy by inhibition of the ubiquitin-proteasome system
-
Meiners S., Dreger H., Fechner M., Bieler S., Rother W., Gunther C., Baumann G., Stangl V., and Stangl K. Suppression of cardiomyocyte hypertrophy by inhibition of the ubiquitin-proteasome system. Hypertension 51 (2008) 302-308
-
(2008)
Hypertension
, vol.51
, pp. 302-308
-
-
Meiners, S.1
Dreger, H.2
Fechner, M.3
Bieler, S.4
Rother, W.5
Gunther, C.6
Baumann, G.7
Stangl, V.8
Stangl, K.9
-
39
-
-
39149097822
-
Proteasome inhibition promotes regression of left ventricular hypertrophy
-
These data demonstrate that proteasome inhibition prevents the development of hypertrophy and promotes the regression of existing hypertrophy in a murine model of agonist-induced cardiac hypertrophy.
-
Stansfield W.E., Tang R.H., Moss N.C., Baldwin A.S., Willis M.S., and Selzman C.H. Proteasome inhibition promotes regression of left ventricular hypertrophy. Am J Physiol Heart Circ Physiol 294 (2008) H645-H650. These data demonstrate that proteasome inhibition prevents the development of hypertrophy and promotes the regression of existing hypertrophy in a murine model of agonist-induced cardiac hypertrophy.
-
(2008)
Am J Physiol Heart Circ Physiol
, vol.294
-
-
Stansfield, W.E.1
Tang, R.H.2
Moss, N.C.3
Baldwin, A.S.4
Willis, M.S.5
Selzman, C.H.6
-
40
-
-
57049121279
-
Proteasome inhibition decreases cardiac remodeling after initiation of pressure overload
-
Hedhli N., Lizano P., Hong C., Fritzky L.F., Dhar S.K., Liu H., Tian Y., Gao S., Madura K., Vatner S.F., et al. Proteasome inhibition decreases cardiac remodeling after initiation of pressure overload. Am J Physiol Heart Circ Physiol 295 (2008) H1385-H1393
-
(2008)
Am J Physiol Heart Circ Physiol
, vol.295
-
-
Hedhli, N.1
Lizano, P.2
Hong, C.3
Fritzky, L.F.4
Dhar, S.K.5
Liu, H.6
Tian, Y.7
Gao, S.8
Madura, K.9
Vatner, S.F.10
-
41
-
-
49849105825
-
Proteasome inhibition attenuates coxsackievirus-induced myocardial damage in mice
-
Gao G., Zhang J., Si X., Wong J., Cheung C., McManus B., and Luo H. Proteasome inhibition attenuates coxsackievirus-induced myocardial damage in mice. Am J Physiol Heart Circ Physiol 295 (2008) H401-H408
-
(2008)
Am J Physiol Heart Circ Physiol
, vol.295
-
-
Gao, G.1
Zhang, J.2
Si, X.3
Wong, J.4
Cheung, C.5
McManus, B.6
Luo, H.7
-
42
-
-
43749098729
-
Proteasome inhibition suppresses essential immune functions of human CD4+ T cells
-
Berges C., Haberstock H., Fuchs D., Miltz M., Sadeghi M., Opelz G., Daniel V., and Naujokat C. Proteasome inhibition suppresses essential immune functions of human CD4+ T cells. Immunology 124 (2008) 234-246
-
(2008)
Immunology
, vol.124
, pp. 234-246
-
-
Berges, C.1
Haberstock, H.2
Fuchs, D.3
Miltz, M.4
Sadeghi, M.5
Opelz, G.6
Daniel, V.7
Naujokat, C.8
-
43
-
-
0034902852
-
A proteasome inhibitor effectively prevents mouse heart allograft rejection
-
Luo H., Wu Y., Qi S., Wan X., Chen H., and Wu J. A proteasome inhibitor effectively prevents mouse heart allograft rejection. Transplantation 72 (2001) 196-202
-
(2001)
Transplantation
, vol.72
, pp. 196-202
-
-
Luo, H.1
Wu, Y.2
Qi, S.3
Wan, X.4
Chen, H.5
Wu, J.6
-
44
-
-
0037192316
-
Ubiquitin-proteasome pathway as a new target for the prevention of restenosis
-
Meiners S., Laule M., Rother W., Guenther C., Prauka I., Muschick P., Baumann G., Kloetzel P.M., and Stangl K. Ubiquitin-proteasome pathway as a new target for the prevention of restenosis. Circulation 105 (2002) 483-489
-
(2002)
Circulation
, vol.105
, pp. 483-489
-
-
Meiners, S.1
Laule, M.2
Rother, W.3
Guenther, C.4
Prauka, I.5
Muschick, P.6
Baumann, G.7
Kloetzel, P.M.8
Stangl, K.9
-
45
-
-
34948873055
-
The proteasome inhibitor lactacystin attenuates growth and migration of vascular smooth muscle cells and limits the response to arterial injury
-
Barringhaus K.G., and Matsumura M.E. The proteasome inhibitor lactacystin attenuates growth and migration of vascular smooth muscle cells and limits the response to arterial injury. Exp Clin Cardiol 12 (2007) 119-124
-
(2007)
Exp Clin Cardiol
, vol.12
, pp. 119-124
-
-
Barringhaus, K.G.1
Matsumura, M.E.2
-
46
-
-
10744223540
-
Long-term up-regulation of eNOS and improvement of endothelial function by inhibition of the ubiquitin-proteasome pathway
-
Stangl V., Lorenz M., Meiners S., Ludwig A., Bartsch C., Moobed M., Vietzke A., Kinkel H.T., Baumann G., and Stangl K. Long-term up-regulation of eNOS and improvement of endothelial function by inhibition of the ubiquitin-proteasome pathway. FASEB J 18 (2004) 272-279
-
(2004)
FASEB J
, vol.18
, pp. 272-279
-
-
Stangl, V.1
Lorenz, M.2
Meiners, S.3
Ludwig, A.4
Bartsch, C.5
Moobed, M.6
Vietzke, A.7
Kinkel, H.T.8
Baumann, G.9
Stangl, K.10
-
47
-
-
34548134851
-
Proteasome-dependent degradation of guanosine 5'-triphosphate cyclohydrolase I causes tetrahydrobiopterin deficiency in diabetes mellitus
-
The authors demonstrate that diabetes induces tetrahydrobiopterin deficiency by increasing proteasome-dependent degradation of guanosine 5'-triphosphate cyclohydrolase I, a rate-limiting enzyme in the synthesis of tetrahydrobiopterin, thus causing impaired endothelial nitric oxide synthase activity and endothelial dysfunction.
-
Xu J., Wu Y., Song P., Zhang M., Wang S., and Zou M.H. Proteasome-dependent degradation of guanosine 5'-triphosphate cyclohydrolase I causes tetrahydrobiopterin deficiency in diabetes mellitus. Circulation 116 (2007) 944-953. The authors demonstrate that diabetes induces tetrahydrobiopterin deficiency by increasing proteasome-dependent degradation of guanosine 5'-triphosphate cyclohydrolase I, a rate-limiting enzyme in the synthesis of tetrahydrobiopterin, thus causing impaired endothelial nitric oxide synthase activity and endothelial dysfunction.
-
(2007)
Circulation
, vol.116
, pp. 944-953
-
-
Xu, J.1
Wu, Y.2
Song, P.3
Zhang, M.4
Wang, S.5
Zou, M.H.6
-
48
-
-
33744978316
-
Increased activity of the ubiquitin-proteasome system in patients with symptomatic carotid disease is associated with enhanced inflammation and may destabilize the atherosclerotic plaque: effects of rosiglitazone treatment
-
Marfella R., D'Amico M., Di Filippo C., Baldi A., Siniscalchi M., Sasso F.C., Portoghese M., Carbonara O., Crescenzi B., Sangiuolo P., et al. Increased activity of the ubiquitin-proteasome system in patients with symptomatic carotid disease is associated with enhanced inflammation and may destabilize the atherosclerotic plaque: effects of rosiglitazone treatment. J Am Coll Cardiol 47 (2006) 2444-2455
-
(2006)
J Am Coll Cardiol
, vol.47
, pp. 2444-2455
-
-
Marfella, R.1
D'Amico, M.2
Di Filippo, C.3
Baldi, A.4
Siniscalchi, M.5
Sasso, F.C.6
Portoghese, M.7
Carbonara, O.8
Crescenzi, B.9
Sangiuolo, P.10
-
49
-
-
33644787193
-
The ubiquitin-proteasome system and inflammatory activity in diabetic atherosclerotic plaques: effects of rosiglitazone treatment
-
Marfella R., D'Amico M., Esposito K., Baldi A., Di Filippo C., Siniscalchi M., Sasso F.C., Portoghese M., Cirillo F., Cacciapuoti F., et al. The ubiquitin-proteasome system and inflammatory activity in diabetic atherosclerotic plaques: effects of rosiglitazone treatment. Diabetes 55 (2006) 622-632
-
(2006)
Diabetes
, vol.55
, pp. 622-632
-
-
Marfella, R.1
D'Amico, M.2
Esposito, K.3
Baldi, A.4
Di Filippo, C.5
Siniscalchi, M.6
Sasso, F.C.7
Portoghese, M.8
Cirillo, F.9
Cacciapuoti, F.10
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