STAT3 single-nucleotide polymorphisms and STAT3 mutations associated with hyper-IgE syndrome are not responsible for increased serum IgE serum levels in asthma families

European Journal of Human Genetics

Volumn 17, Issue 3, 2009, Pages 352-356

  Wjst, Matthias ^a,b   Lichtner, Peter ^a   Meitinger, Thomas ^a,c   Grimbacher, Bodo ^d  

^a INSTITUTE OF EPIDEMIOLOGY   (Germany)

^b INSTITUTE FOR RENEWABLE ENERGY   (Italy)

^c TECHNICAL UNIVERSITY OF MUNICH   (Germany)

^d ROYAL FREE HOSPITAL   (United Kingdom)

Author keywords

[No Author keywords available]

Indexed keywords

DNA; IMMUNOGLOBULIN E; STAT3 PROTEIN;

5' UNTRANSLATED REGION; ALLERGIC RHINITIS; ARTICLE; ASTHMA; CONTROLLED STUDY; ECZEMA; EOSINOPHIL COUNT; FAMILY; FEMALE; GENE MUTATION; GENETIC ASSOCIATION; GENETIC VARIABILITY; GENOTYPE; HAPLOTYPE; HUMAN; HUMAN CELL; HYPER IGE SYNDROME; IMMUNOGLOBULIN BLOOD LEVEL; LUNG FUNCTION; MAJOR CLINICAL STUDY; MALE; MATRIX ASSISTED LASER DESORPTION IONIZATION TIME OF FLIGHT MASS SPECTROMETRY; PRIORITY JOURNAL; PROMOTER REGION; SCHOOL CHILD; SINGLE NUCLEOTIDE POLYMORPHISM;

ASTHMA; CHILD; ECZEMA; FAMILY; HUMANS; IMMUNOGLOBULIN E; JOB'S SYNDROME; MUTATION; POLYMORPHISM, SINGLE NUCLEOTIDE; SPECTROMETRY, MASS, MATRIX-ASSISTED LASER DESORPTION-IONIZATION; STAT3 TRANSCRIPTION FACTOR;

EID: 60749113901     PISSN: 10184813     EISSN: 14765438     Source Type: Journal    
DOI: 10.1038/ejhg.2008.169     Document Type: Article

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