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Volumn 23, Issue 2, 2009, Pages 417-419
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Redundant function of retinoic acid receptor isoforms in leukemogenesis unravels a prominent function of genome topology and architecture in the selection of mutagenic events in cancer
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Author keywords
[No Author keywords available]
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Indexed keywords
RETINOIC ACID RECEPTOR ALPHA;
RETINOIC ACID RECEPTOR BETA;
RETINOIC ACID RECEPTOR GAMMA;
CANCER GENETICS;
CELL SELECTION;
CHROMOSOME 15;
CHROMOSOME 17;
CHROMOSOME TRANSLOCATION;
DISEASE MODEL;
FLUORESCENCE ACTIVATED CELL SORTING;
GENOME;
HEMATOPOIETIC STEM CELL;
IMMUNOPHENOTYPING;
LETTER;
LEUKEMOGENESIS;
MUTAGENESIS;
NONHUMAN;
PHENOTYPE;
PRIORITY JOURNAL;
PROMYELOCYTIC LEUKEMIA;
RECEPTOR INTRINSIC ACTIVITY;
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EID: 60149085217
PISSN: 08876924
EISSN: 14765551
Source Type: Journal
DOI: 10.1038/leu.2008.205 Document Type: Letter |
Times cited : (15)
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References (8)
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