CELF-mediated alternative splicing is required for cardiac function during early, but not later, postnatal life

Journal of Molecular and Cellular Cardiology

Volumn 46, Issue 3, 2009, Pages 395-404

  Terenzi, Fulvia ^a   Brimacombe, Kyle R ^a   Penn, Marc S ^a   Ladd, Andrea N ^a  

^a LERNER RESEARCH INSTITUTE   (United States)

Author keywords

Alternative splicing; Cardiomyopathy; CELF proteins; Development; Postnatal remodeling; Transgenic mice

Indexed keywords

BINDING PROTEIN; RNA BINDING PROTEIN;

ALTERNATIVE RNA SPLICING; ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; CARDIOMYOPATHY; CONTROLLED STUDY; FEMALE; GENE EXPRESSION; GENETICS; GENOTYPE; HEART DILATATION; HEART FUNCTION; HEART SIZE; MALE; MOUSE; MOUSE STRAIN; NONHUMAN; PERINATAL PERIOD; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN FUNCTION; TISSUE REPAIR; TRANSGENIC MOUSE;

AGING; ALTERNATIVE SPLICING; ANIMALS; CARDIOMYOPATHIES; CCAAT-ENHANCER-BINDING PROTEIN-DELTA; FEMALE; HEART; MALE; MICE; MICE, TRANSGENIC; MUSCLE PROTEINS; MYOCARDIUM; ORGAN SIZE; ORGAN SPECIFICITY;

MUS; MUS MUSCULUS;

EID: 59149085793     PISSN: 00222828     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.yjmcc.2008.10.030     Document Type: Article

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