Activation of the aryl hydrocarbon receptor by TCDD inhibits senescence: A tumor promoting event?

Biochemical Pharmacology

Volumn 77, Issue 4, 2009, Pages 681-688

  Ray, S ^b   Swanson, H I ^a  

^a UNIVERSITY OF KENTUCKY   (United States)

^b BIOGEN   (United States)

Author keywords

2,3,7,8 Tetrachlorodibenzo p dioxin; Aryl hydrocarbon receptor; Keratinocytes; Senescence; Tumorigenesis

Indexed keywords

2,3,7,8 TETRACHLORODIBENZO PARA DIOXIN; AROMATIC HYDROCARBON RECEPTOR;

APOPTOSIS; CELL AGING; CELL CYCLE ARREST; CELL DIFFERENTIATION; CHEMICAL CARCINOGENESIS; HUMAN; KERATINOCYTE; NONHUMAN; PRIORITY JOURNAL; REVIEW; SIGNAL TRANSDUCTION; SKIN CANCER; SKIN CARCINOGENESIS;

AGING; ANIMALS; APOPTOSIS; ENVIRONMENTAL POLLUTANTS; HUMANS; NEOPLASMS; RECEPTORS, ARYL HYDROCARBON; TETRACHLORODIBENZODIOXIN;

EID: 59149083365     PISSN: 00062952     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.bcp.2008.11.022     Document Type: Review

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