A molecular correlate of clinicopathology in transthyretin amyloidosis

Journal of Pathology

Volumn 217, Issue 1, 2009, Pages 1-3

  Pepys, Mark B ^a,b  

^a ROYAL FREE AND UNIVERSITY COLLEGE MEDICAL SCHOOL   (United Kingdom)

^b UNIVERSITY COLLEGE LONDON   (United Kingdom)

Author keywords

Amyloidosis; Fibril; Transthyretin

Indexed keywords

AMYLOID; METHIONINE; PREALBUMIN; VALINE;

AMYLOIDOSIS; CELL ACTIVITY; GENE MUTATION; HISTOPATHOLOGY; HUMAN; NOTE; PRIORITY JOURNAL; PROTEIN FOLDING; PROTEIN VARIANT; WILD TYPE;

AMYLOID; AMYLOIDOSIS; HUMANS; MUTATION; PREALBUMIN;

EID: 58449110805     PISSN: 00223417     EISSN: 10969896     Source Type: Journal    
DOI: 10.1002/path.2467     Document Type: Note

References (4)

1. Pepys MB, Hawkins PN. Amyloidosis. In Oxford Textbook of Medicine (4th edn), vol 2, Warrell DA, Cox TM, Firth JD, Benz EJ Jr (eds). Oxford University Press: Oxford, 2003; 162-173.
2. Pepys MB. Amyloidosis. Annu Rev Med 2006;57:223-241.
3. Puchtler H, Sweat F, Levine M. On the binding of Congo red by amyloid. J Histochem Cytochem 1962;10:355-364.
4. Ihse E, Ybo A, Suhr OB, Lindqvist P, Backman C, Westermark P. Amyloid fibril composition is related to the phenotype of hereditary transthyretin V30M amyloidosis. J Pathol 2008;216:253 - 261.