Human and primate tumour viruses use PDZ binding as an evolutionarily conserved mechanism of targeting cell polarity regulators

Oncogene

Volumn 28, Issue 1, 2009, Pages 1-8

  Tomaic V ^a   Gardiol, D ^a   Massimi, P ^a   Ozbun, M ^b   Myers, M ^a   Banks, L ^a  

^a INTERNATIONAL CENTRE FOR GENETIC ENGINEERING AND BIOTECHNOLOGY   (Italy)

^b UNIVERSITY OF NEW MEXICO SCHOOL OF MEDICINE   (United States)

Author keywords

Oncoproteins; Papillomavirus; Par3; PDZ domains

Indexed keywords

ONCOPROTEIN; PDZ PROTEIN; PROTEASOME;

ANIMAL CELL; ANIMAL EXPERIMENT; ARTICLE; BINDING AFFINITY; CELL INTERACTION; CELL POLARITY; CONTROLLED STUDY; DEGRADATION; NONHUMAN; PAPILLOMA VIRUS; PRIMATE; PRIORITY JOURNAL; PROTEIN TARGETING; TUMOR VIRUS;

ADAPTOR PROTEINS, SIGNAL TRANSDUCING; AMINO ACID MOTIFS; AMINO ACID SEQUENCE; ANIMALS; CELL CYCLE PROTEINS; CELL LINE; CELL POLARITY; CONSERVED SEQUENCE; EVOLUTION, MOLECULAR; HUMANS; MACACA MULATTA; MEMBRANE PROTEINS; MOLECULAR SEQUENCE DATA; ONCOGENE PROTEINS, VIRAL; ONCOGENIC VIRUSES; PAPILLOMAVIRIDAE; PDZ DOMAINS; TUMOR SUPPRESSOR PROTEINS;

HUMAN PAPILLOMAVIRUS; MACACA MULATTA; PAPILLOMAVIRIDAE; PRIMATES;

EID: 58149359224     PISSN: 09509232     EISSN: 14765594     Source Type: Journal    
DOI: 10.1038/onc.2008.365     Document Type: Article

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